The effect of alcohol and hydrogen peroxide on liver hepcidin gene expression in mice lacking antioxidant enzymes, glutathione peroxidase-1 or catalase

Biomolecules

Volumn 5, Issue 2, 2015, Pages 793-807

  Harrison Findik, Duygu Dee ^a   Lu, Sizhao ^a  

^a UNIVERSITY OF NEBRASKA MEDICAL CENTER   (United States)

Author keywords

Alcoholic liver disease; CHOP; Endoplasmic reticulum stress; Hamp; Hepatocyte; Iron; Oxidative stress

Indexed keywords

ALCOHOL; CATALASE; CYTOCHROME P450 2E1; GLUTATHIONE PEROXIDASE; GROWTH ARREST AND DNA DAMAGE INDUCIBLE PROTEIN 153; HEPCIDIN; HISTONE DEACETYLASE; HYDROGEN PEROXIDE; X BOX BINDING PROTEIN 1; DDIT3 PROTEIN, MOUSE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CELL VIABILITY; CHROMATIN IMMUNOPRECIPITATION; CONTROLLED STUDY; ENDOPLASMIC RETICULUM STRESS; GENE EXPRESSION; GENE SILENCING; MOUSE; NONHUMAN; OXIDATIVE STRESS; REAL TIME POLYMERASE CHAIN REACTION; SPECTROPHOTOMETRY; UPREGULATION; WESTERN BLOTTING; ANIMAL; C57BL MOUSE; CELL CULTURE; DEFICIENCY; DRUG EFFECTS; GENETICS; LIVER; METABOLISM;

MUS;

ANIMALS; CATALASE; CELLS, CULTURED; ETHANOL; GLUTATHIONE PEROXIDASE; HEPCIDINS; HYDROGEN PEROXIDE; LIVER; MICE; MICE, INBRED C57BL; TRANSCRIPTION FACTOR CHOP;

EID: 85012054090     PISSN: None     EISSN: 2218273X     Source Type: Journal    
DOI: 10.3390/biom5020793     Document Type: Article

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