Caspase-Cleaved Tau Impairs Mitochondrial Dynamics in Alzheimer’s Disease

Molecular Neurobiology

Volumn 55, Issue 2, 2018, Pages 1004-1018

  Pérez, María José ^a   Vergara Pulgar, Katiana ^a   Jara, Claudia ^a   Cabezas Opazo, Fabian ^a   Quintanilla, Rodrigo A ^a,b  

^a UNIVERSIDAD AUTÓNOMA DE CHILE   (Chile)

^b Centro de Investigación y Estudio del Consumo de Alcohol en Adolescentes   (Chile)

Author keywords

Alzheimer s disease; Mitochondria; Neurodegeneration; Opa1; Tau

Indexed keywords

AMYLOID BETA PROTEIN; CASPASE; GREEN FLUORESCENT PROTEIN; MITOCHONDRIAL PROTEIN; OPTIC ATROPHY PROTEIN 1; TAU PROTEIN; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; ANIMAL CELL; ARTICLE; BRAIN CELL; CELL SIZE; CELL VIABILITY; CONTROLLED STUDY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; DOWN REGULATION; IMMORTALIZED CELL LINE; MITOCHONDRIAL MEMBRANE POTENTIAL; MOUSE; NONHUMAN; PROTEIN CLEAVAGE; PROTEIN EXPRESSION; PROTEIN MODIFICATION; PROTEIN PHOSPHORYLATION; ANIMAL; BRAIN; GENETICS; KNOCKOUT MOUSE; METABOLISM; MITOCHONDRIAL DYNAMICS; MITOCHONDRION; NERVE CELL; PHOSPHORYLATION; PHYSIOLOGY;

ALZHEIMER DISEASE; ANIMALS; BRAIN; GREEN FLUORESCENT PROTEINS; MEMBRANE POTENTIAL, MITOCHONDRIAL; MICE; MICE, KNOCKOUT; MITOCHONDRIA; MITOCHONDRIAL DYNAMICS; NEURONS; PHOSPHORYLATION; TAU PROTEINS;

EID: 85010711827     PISSN: 08937648     EISSN: 15591182     Source Type: Journal    
DOI: 10.1007/s12035-017-0385-x     Document Type: Article

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