The transcription factor PU.1 promotes alternative macrophage polarization and asthmatic airway inflammation

Journal of Molecular Cell Biology

Volumn 7, Issue 6, 2015, Pages 557-567

  Qian, Feng ^a,b   Deng, Jing ^b   Lee, Yong Gyu ^b   Zhu, Jimmy ^c   Karpurapu, Manjula ^b   Chung, Sangwoon ^b   Zheng, Jun Nian ^d   Xiao, Lei ^e   Park, Gye Young ^e   Christman, John W ^b  

^a SHANGHAI JIAO TONG UNIVERSITY   (China)

^b OHIO STATE UNIVERSITY   (United States)

^c UNIVERSITY OF ILLINOIS   (United States)

^d XUZHOU MEDICAL UNIVERSITY   (China)

^e UNIVERSITY OF ILLINOIS AT CHICAGO   (United States)

Author keywords

Alternatively activated macrophage; Asthmatic inflammation; IL 4; PU.1; Th2

Indexed keywords

ALLERGEN; CELL PROTEIN; CHITINASE 3 LIKE 3; DUST MITE; IMMUNOGLOBULIN E; INTERLEUKIN 4; MESSENGER RNA; RESISTIN LIKE MOLECULE ALPHA 1; STAT6 PROTEIN; TRANSCRIPTION FACTOR PU 1; UNCLASSIFIED DRUG; BETA N ACETYLHEXOSAMINIDASE; CHI3L3 PROTEIN, MOUSE; LECTIN; ONCOPROTEIN; PROTO-ONCOGENE PROTEIN SPI-1; RETNLA PROTEIN, MOUSE; SIGNAL PEPTIDE; TRANSACTIVATOR PROTEIN;

ADOPTIVE TRANSFER; ALTERNATIVE MACROPHAGE POLARIZATION; ALTERNATIVELY ACTIVATED MACROPHAGE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ASPERGILLUS; ASTHMA; ASTHMATIC AIRWAY INFLAMMATION; CELL HYPERPLASIA; CELL INFILTRATION; CELLULAR IMMUNITY; CELLULAR PARAMETERS; CONTROLLED STUDY; DISEASE ASSOCIATION; DISEASE COURSE; EMBRYO; EOSINOPHIL; GENE EXPRESSION; GOBLET CELL; HISTOPATHOLOGY; IN VITRO STUDY; INFLAMMATION; MACROPHAGE; MOLECULAR DYNAMICS; MOUSE; NONHUMAN; PATHOGENESIS; PHENOTYPE; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; PROTEIN SYNTHESIS; PROVOCATION TEST; RAGWEED; WILD TYPE; ANIMAL; CYTOLOGY; GENETICS; HYPERSENSITIVITY; IMMUNOLOGY; LUNG; LUNG ALVEOLUS; MACROPHAGE ACTIVATION; METABOLISM; NEUTROPHIL CHEMOTAXIS; PATHOLOGY; TH2 CELL; TRANSGENIC MOUSE;

ADOPTIVE TRANSFER; ALLERGENS; ANIMALS; ASTHMA; BETA-N-ACETYLHEXOSAMINIDASES; CHEMOTAXIS, LEUKOCYTE; EOSINOPHILS; HYPERSENSITIVITY; INFLAMMATION; INTERCELLULAR SIGNALING PEPTIDES AND PROTEINS; INTERLEUKIN-4; LECTINS; LUNG; MACROPHAGE ACTIVATION; MACROPHAGES; MICE; MICE, TRANSGENIC; PROTO-ONCOGENE PROTEINS; PULMONARY ALVEOLI; TH2 CELLS; TRANS-ACTIVATORS;

EID: 84975784690     PISSN: 16742788     EISSN: 17594685     Source Type: Journal    
DOI: 10.1093/jmcb/mjv042     Document Type: Article

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