Alternative activation is an innate response to injury that requires CD4+ T cells to be sustained during chronic infection

Journal of Immunology

Volumn 179, Issue 6, 2007, Pages 3926-3936

  Loke, P'Ng ^a   Gallagher, Iain ^b   Nair, Meera G ^b,f   Zang, Xingxing ^c   Brombacher, Frank ^d   Mohrs, Markus ^e   Allison, James P ^c   Allen, Judith E ^b  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF EDINBURGH   (United Kingdom)

^c MEMORIAL SLOAN KETTERING CANCER CENTER   (United States)

^d UNIVERSITY OF CAPE TOWN   (South Africa)

^e TRUDEAU INSTITUTE   (United States)

^f UNIVERSITY OF PENNSYLVANIA SCHOOL OF VETERINARY MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ARGINASE 1; INTERLEUKIN 13; INTERLEUKIN 4; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 2; ARGINASE; BETA N ACETYLHEXOSAMINIDASE; CHI3L3 PROTEIN, MOUSE; LECTIN; NERVE GROWTH FACTOR; PROTEIN; RESISTIN LIKE ALPHA, MOUSE; RESISTIN-LIKE ALPHA, MOUSE; UNCLASSIFIED DRUG;

ADAPTIVE IMMUNITY; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; B LYMPHOCYTE; BRUGIA MALAYI; CD4+ T LYMPHOCYTE; CONTROLLED STUDY; EOSINOPHILIA; FEMALE; INNATE IMMUNITY; MACROPHAGE ACTIVATION; MALE; MOUSE; NEMATODIASIS; NEUTROPHILIA; NONHUMAN; PHAGOCYTOSIS; PRIORITY JOURNAL; T LYMPHOCYTE; TH2 CELL; TISSUE INJURY; UPREGULATION; ACTIVE IMMUNIZATION; ANIMAL; BIOSYNTHESIS; C57BL MOUSE; CELL MOTION; CHRONIC DISEASE; DRUG ANTAGONISM; FILARIASIS; GENETICS; IMMUNOLOGY; METABOLISM; MOUSE MUTANT; NEUTROPHIL; PARASITOLOGY; PATHOLOGY;

ANIMALS; ARGINASE; BETA-N-ACETYLHEXOSAMINIDASE; BRUGIA MALAYI; CD4-POSITIVE T-LYMPHOCYTES; CELL MOVEMENT; CHRONIC DISEASE; FEMALE; FILARIASIS; IMMUNITY, ACTIVE; LECTINS; MACROPHAGE ACTIVATION; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NERVE GROWTH FACTOR; NEUTROPHILS; PROTEINS;

EID: 35748964164     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.179.6.3926     Document Type: Article

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