CHOP contributes to, but is not the only mediator of, IAPP induced β-cell apoptosis

Molecular Endocrinology

Volumn 30, Issue 4, 2016, Pages 446-454

  Gurlo, T ^a   Rivera, J F ^a   Butler, A E ^a   Cory, M ^a   Hoang, J ^a   Costes, S ^a   Butler, Peter C ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ACTIVATING TRANSCRIPTION FACTOR 4; AMYLIN; BIM PROTEIN; CCAAT ENHANCER BINDING PROTEIN HOMOLOGOUS PROTEIN; INOSITOL REQUIRING ENZYME 1 PHOSPHORYLATED TUMOR NECROSIS FACTOR RECEPTOR ASSOCIATED FACTOR 2; PROTEIN; PROTEIN BCL 2; STRESS ACTIVATED PROTEIN KINASE; UNCLASSIFIED DRUG; DDIT3 PROTEIN, MOUSE; GROWTH ARREST AND DNA DAMAGE INDUCIBLE PROTEIN 153;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; AUTOPHAGY; CELL PROLIFERATION; CONTROLLED STUDY; CYTOTOXICITY; DIABETES MELLITUS; ENDOPLASMIC RETICULUM STRESS; GENE DELETION; LYSOSOME; MALE; MOLECULAR BIOLOGY; MOUSE; NONHUMAN; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FAMILY; ANIMAL; C57BL MOUSE; GENETICS; HUMAN; KNOCKOUT MOUSE; METABOLISM; NON INSULIN DEPENDENT DIABETES MELLITUS; PATHOLOGY; PHYSIOLOGY; SIGNAL TRANSDUCTION;

ANIMALS; APOPTOSIS; DIABETES MELLITUS, TYPE 2; ENDOPLASMIC RETICULUM STRESS; HUMANS; INSULIN-SECRETING CELLS; ISLET AMYLOID POLYPEPTIDE; MALE; MICE, INBRED C57BL; MICE, KNOCKOUT; SIGNAL TRANSDUCTION; TRANSCRIPTION FACTOR CHOP;

EID: 84964294550     PISSN: 08888809     EISSN: 19449917     Source Type: Journal    
DOI: 10.1210/me.2015-1255     Document Type: Article

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