Disruption of ah receptor signaling during mouse development leads to abnormal cardiac structure and function in the adult

PLoS ONE

Volumn 10, Issue 11, 2015, Pages

  Carreira, Vinicius S ^a   Fan, Yunxia ^a   Kurita, Hisaka ^a   Wang, Qin ^a   Ko, Chia I ^a   Naticchioni, Mindi ^a   Jiang, Min ^a   Koch, Sheryl ^a   Zhang, Xiang ^a   Biesiada, Jacek ^a   Medvedovic, Mario ^a   Xia, Ying ^a   Rubinstein, Jack ^a   Puga, Alvaro ^a  

^a UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN; AROMATIC HYDROCARBON RECEPTOR;

ADULT; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CALCIUM SIGNALING; CONGENITAL HEART DISEASE; CONTROLLED STUDY; DISEASE SEVERITY; EMBRYO DEVELOPMENT; EXERCISE TOLERANCE; FEMALE; HEART LEFT VENTRICLE EJECTION FRACTION; HEART LEFT VENTRICLE MASS; HEART LEFT VENTRICLE VOLUME; HEART MITOCHONDRION; HEART VENTRICLE HYPERTROPHY; HEART WEIGHT; MALE; MEAN ARTERIAL PRESSURE; MOUSE; NONHUMAN; PRENATAL EXPOSURE; RESTING HEART RATE; SEX DIFFERENCE; SYSTOLIC BLOOD PRESSURE; TRANSCRIPTOMICS; ANIMAL; C57BL MOUSE; CONGENITAL HEART MALFORMATION; GENETICS; HOMEOSTASIS; MATERNAL EXPOSURE; METABOLISM; PATHOPHYSIOLOGY; PREGNANCY; SIGNAL TRANSDUCTION; TRANSGENIC MOUSE;

ANIMALS; FEMALE; HEART DEFECTS, CONGENITAL; HOMEOSTASIS; MATERNAL EXPOSURE; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MITOCHONDRIA, HEART; PREGNANCY; RECEPTORS, ARYL HYDROCARBON; SIGNAL TRANSDUCTION;

EID: 84953331807     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0142440     Document Type: Article

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