Ah receptor signaling controls the expression of cardiac development and homeostasis genes

Toxicological Sciences

Volumn 147, Issue 2, 2015, Pages 425-435

  Carreira, Vinicius S ^a   Fan, Yunxia ^a   Wang, Qing ^a   Zhang, Xiang ^a   Kurita, Hisaka ^a   Ko, Chia I ^a   Naticchioni, Mindi ^a   Jiang, Min ^a   Koch, Sheryl ^a   Medvedovic, Mario ^a   Xia, Ying ^a   Rubinstein, Jack ^a   Puga, Alvaro ^a  

^a UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

Ah receptor; Cardiogenesis; Congenital heart disease; Mitochondrial dysfunction; NKX2 5

Indexed keywords

AROMATIC HYDROCARBON RECEPTOR; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR NKH2.5; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL DIFFERENTIATION; CELL METABOLISM; CITRIC ACID CYCLE; COMPARATIVE STUDY; CONGENITAL HEART DISEASE; CONTROLLED STUDY; DISEASE ASSOCIATION; EMBRYO; EMBRYO DEVELOPMENT; ENERGY BALANCE; ENVIRONMENTAL FACTOR; FEMALE; FETUS HEART; FETUS WEIGHT; GENE CONTROL; GENETIC CODE; GENETIC REGULATION; HEART DEVELOPMENT; HEART EJECTION FRACTION; HEART MITOCHONDRION; HEART RATE; HEART STROKE VOLUME; HOMEOSTASIS; MOUSE; NONHUMAN; OXIDATIVE PHOSPHORYLATION; SIGNAL TRANSDUCTION; TRANSMISSION ELECTRON MICROSCOPY; ANIMAL; C57BL MOUSE; CARDIAC MUSCLE; DRUG EFFECTS; ECHOCARDIOGRAPHY; EMBRYOLOGY; GENETICS; GROWTH, DEVELOPMENT AND AGING; HEART; MALE; METABOLISM; PATHOLOGY; PHYSIOLOGY; ULTRASTRUCTURE;

ANIMALS; ECHOCARDIOGRAPHY; FEMALE; HEART; HOMEOSTASIS; MALE; MICE; MICE, INBRED C57BL; MICROSCOPY, ELECTRON, TRANSMISSION; MITOCHONDRIA, HEART; MYOCARDIUM; RECEPTORS, ARYL HYDROCARBON; SIGNAL TRANSDUCTION;

EID: 84943801549     PISSN: 10966080     EISSN: 10960929     Source Type: Journal    
DOI: 10.1093/toxsci/kfv138     Document Type: Article

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