Prolonged depletion of AH receptor without alteration of receptor mRNA levels after treatment of cells in culture with 2,3,7,8-tetrachlorodibenzo-p-dioxin

Biochemical Pharmacology

Volumn 55, Issue 4, 1998, Pages 489-497

  Giannone, John V ^a   Li, Wei ^a   Probst, Markus ^b   Okey, Allan B ^a  

^a UNIVERSITY OF TORONTO   (Canada)

^b UCLA MEDICAL CENTER   (United States)

Author keywords

2,3,7,8 tetrachlorodibenzo p dioxin; 3 methylcholanthrene; AH receptor; AH receptor nuclear translocator protein (ARNT); Hepa 1 hepatoma cells; TCDD

Indexed keywords

2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN; 3 METHYLCHOLANTHRENE; AROMATIC HYDROCARBON RECEPTOR; CARRIER PROTEIN; DACTINOMYCIN; MESSENGER RNA;

ANIMAL CELL; ARTICLE; CELL CULTURE; CONTROLLED STUDY; GENE EXPRESSION; HEPATOMA CELL; IMMUNOBLOTTING; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION;

ANIMALS; ARYL HYDROCARBON RECEPTOR NUCLEAR TRANSLOCATOR; BASE SEQUENCE; CELL LINE; CELL NUCLEUS; CYTOSOL; DACTINOMYCIN; DNA PRIMERS; DNA-BINDING PROTEINS; KINETICS; METHYLCHOLANTHRENE; MICE; POLYMERASE CHAIN REACTION; RECEPTORS, ARYL HYDROCARBON; RNA, MESSENGER; TETRACHLORODIBENZODIOXIN; TRANSCRIPTION FACTORS; TRANSCRIPTION, GENETIC;

EID: 0032519879     PISSN: 00062952     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0006-2952(97)00493-0     Document Type: Article

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