PI3K and AKT: Unfaithful partners in cancer

International Journal of Molecular Sciences

Volumn 16, Issue 9, 2015, Pages 21138-21152

  Faes, Seraina ^a   Dormond, Olivier ^a  

^a LAUSANNE UNIVERSITY HOSPITAL   (Switzerland)

Author keywords

AKT; Cancer; PI3K; Signaling; Therapies

Indexed keywords

IDELALISIB; INTERLEUKIN 8; JANUS KINASE 2; PHOSPHATIDYLINOSITOL 3 KINASE; PHOSPHATIDYLINOSITOL 3 KINASE INHIBITOR; PROTEIN KINASE B; PROTEIN KINASE B INHIBITOR; PROTEIN P110; TRANSCRIPTION FACTOR FKHR; ANTINEOPLASTIC AGENT; PROTEIN KINASE INHIBITOR;

ANTINEOPLASTIC ACTIVITY; CARCINOGENESIS; CLINICAL TRIAL (TOPIC); DNA FLANKING REGION; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME SUBUNIT; GENE MUTATION; HUMAN; NEGATIVE FEEDBACK; NEOPLASM; PHASE 2 CLINICAL TRIAL (TOPIC); PHASE 3 CLINICAL TRIAL (TOPIC); PHOSPHORYLATION; REVIEW; SIGNAL TRANSDUCTION; TUMOR GROWTH; TUMOR MICROENVIRONMENT; METABOLISM; NEOPLASMS;

ANTINEOPLASTIC AGENTS; HUMANS; NEOPLASMS; PHOSPHATIDYLINOSITOL 3-KINASE; PROTEIN KINASE INHIBITORS; PROTO-ONCOGENE PROTEINS C-AKT; SIGNAL TRANSDUCTION;

EID: 84941011455     PISSN: 16616596     EISSN: 14220067     Source Type: Journal    
DOI: 10.3390/ijms160921138     Document Type: Review

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