-
1
-
-
0028085454
-
Alternative splicing introduces a nuclear localization signal that targets multifunctional CaM kinase to the nucleus
-
Srinivasan M., Edman C.F., Schulman H. Alternative splicing introduces a nuclear localization signal that targets multifunctional CaM kinase to the nucleus. J Cell Biol 1994, 126:839-852.
-
(1994)
J Cell Biol
, vol.126
, pp. 839-852
-
-
Srinivasan, M.1
Edman, C.F.2
Schulman, H.3
-
2
-
-
84856087553
-
Location matters: clarifying the concept of nuclear and cytosolic CaMKII subtypes
-
Mishra S., Gray C.B., Miyamoto S., Bers D.M., Brown J.H. Location matters: clarifying the concept of nuclear and cytosolic CaMKII subtypes. Circ Res 2011, 109:1354-1362.
-
(2011)
Circ Res
, vol.109
, pp. 1354-1362
-
-
Mishra, S.1
Gray, C.B.2
Miyamoto, S.3
Bers, D.M.4
Brown, J.H.5
-
3
-
-
84862296973
-
Calmodulin-dependent protein kinase II: linking heart failure and arrhythmias
-
Swaminathan P.D., Purohit A., Hund T.J., Anderson M.E. Calmodulin-dependent protein kinase II: linking heart failure and arrhythmias. Circ Res 2012, 110:1661-1677.
-
(2012)
Circ Res
, vol.110
, pp. 1661-1677
-
-
Swaminathan, P.D.1
Purohit, A.2
Hund, T.J.3
Anderson, M.E.4
-
5
-
-
0028306195
-
Dual role of calmodulin in autophosphorylation of multifunctional CaM kinase may underlie decoding of calcium signals
-
Hanson P.I., Meyer T., Stryer L., Schulman H. Dual role of calmodulin in autophosphorylation of multifunctional CaM kinase may underlie decoding of calcium signals. Neuron 1994, 12:943-956.
-
(1994)
Neuron
, vol.12
, pp. 943-956
-
-
Hanson, P.I.1
Meyer, T.2
Stryer, L.3
Schulman, H.4
-
6
-
-
42949085382
-
A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation
-
Erickson J.R., Joiner M.L., Guan X., Kutschke W., Yang J., Oddis C.V., et al. A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation. Cell 2008, 133:462-474.
-
(2008)
Cell
, vol.133
, pp. 462-474
-
-
Erickson, J.R.1
Joiner, M.L.2
Guan, X.3
Kutschke, W.4
Yang, J.5
Oddis, C.V.6
-
7
-
-
84885863770
-
Diabetic hyperglycaemia activates CaMKII and arrhythmias by O-linked glycosylation
-
Erickson J.R., Pereira L., Wang L., Han G., Ferguson A., Dao K., et al. Diabetic hyperglycaemia activates CaMKII and arrhythmias by O-linked glycosylation. Nature 2013, 502:372-376.
-
(2013)
Nature
, vol.502
, pp. 372-376
-
-
Erickson, J.R.1
Pereira, L.2
Wang, L.3
Han, G.4
Ferguson, A.5
Dao, K.6
-
8
-
-
70349754717
-
Calcium/calmodulin-dependent kinase II regulation of cardiac ion channels
-
Bers D.M., Grandi E. Calcium/calmodulin-dependent kinase II regulation of cardiac ion channels. J Cardiovasc Pharmacol 2009, 54:180-187.
-
(2009)
J Cardiovasc Pharmacol
, vol.54
, pp. 180-187
-
-
Bers, D.M.1
Grandi, E.2
-
10
-
-
84904723563
-
2+ current facilitation is CaMKII-dependent and has arrhythmogenic consequences
-
2+ current facilitation is CaMKII-dependent and has arrhythmogenic consequences. Front Pharmacol 2014, 5:144.
-
(2014)
Front Pharmacol
, vol.5
, pp. 144
-
-
Bers, D.M.1
Morotti, S.2
-
11
-
-
84897111408
-
Ryanodine receptor- mediated calcium leak drives progressive development of an atrial fibrillation substrate in a transgenic mouse model
-
Li N., Chiang D.Y., Wang S., Wang Q., Sun L., Voigt N., et al. Ryanodine receptor- mediated calcium leak drives progressive development of an atrial fibrillation substrate in a transgenic mouse model. Circulation 2014, 129:1276-1285.
-
(2014)
Circulation
, vol.129
, pp. 1276-1285
-
-
Li, N.1
Chiang, D.Y.2
Wang, S.3
Wang, Q.4
Sun, L.5
Voigt, N.6
-
12
-
-
33845284849
-
Ca/calmodulin-dependent protein kinase II regulates cardiac Na channels
-
Wagner S., Dybkova N., Rasenack E.C., Jacobshagen C., Fabritz L., Kirchhof P., et al. Ca/calmodulin-dependent protein kinase II regulates cardiac Na channels. J Clin Invest 2006, 116:3127-3138.
-
(2006)
J Clin Invest
, vol.116
, pp. 3127-3138
-
-
Wagner, S.1
Dybkova, N.2
Rasenack, E.C.3
Jacobshagen, C.4
Fabritz, L.5
Kirchhof, P.6
-
13
-
-
84886408667
-
BetaIV-Spectrin and CaMKII facilitate Kir6.2 regulation in pancreatic beta cells
-
Kline C.F., Wright P.J., Koval O.M., Zmuda E.J., Johnson B.L., Anderson M.E., et al. betaIV-Spectrin and CaMKII facilitate Kir6.2 regulation in pancreatic beta cells. Proc Natl Acad Sci U S A 2013, 110:17576-17581.
-
(2013)
Proc Natl Acad Sci U S A
, vol.110
, pp. 17576-17581
-
-
Kline, C.F.1
Wright, P.J.2
Koval, O.M.3
Zmuda, E.J.4
Johnson, B.L.5
Anderson, M.E.6
-
14
-
-
84872736412
-
Regulation of cardiac ATP-sensitive potassium channel surface expression by calcium/calmodulin-dependent protein kinase II
-
Sierra A., Zhu Z., Sapay N., Sharotri V., Kline C.F., Luczak E.D., et al. Regulation of cardiac ATP-sensitive potassium channel surface expression by calcium/calmodulin-dependent protein kinase II. J Biol Chem 2013, 288:1568-1581.
-
(2013)
J Biol Chem
, vol.288
, pp. 1568-1581
-
-
Sierra, A.1
Zhu, Z.2
Sapay, N.3
Sharotri, V.4
Kline, C.F.5
Luczak, E.D.6
-
15
-
-
77956528473
-
Ankyrin-B regulates Kir6.2 membrane expression and function in heart
-
Li J., Kline C.F., Hund T.J., Anderson M.E., Mohler P.J. Ankyrin-B regulates Kir6.2 membrane expression and function in heart. J Biol Chem 2010, 285:28723-28730.
-
(2010)
J Biol Chem
, vol.285
, pp. 28723-28730
-
-
Li, J.1
Kline, C.F.2
Hund, T.J.3
Anderson, M.E.4
Mohler, P.J.5
-
16
-
-
77954518589
-
Cardiomyocytes with disrupted CFTR function require CaMKII and Ca(2+)-activated Cl(-) channel activity to maintain contraction rate
-
Sellers Z.M., De Arcangelis V., Xiang Y., Best P.M. Cardiomyocytes with disrupted CFTR function require CaMKII and Ca(2+)-activated Cl(-) channel activity to maintain contraction rate. J Physiol 2010, 588:2417-2429.
-
(2010)
J Physiol
, vol.588
, pp. 2417-2429
-
-
Sellers, Z.M.1
De Arcangelis, V.2
Xiang, Y.3
Best, P.M.4
-
17
-
-
15244350340
-
Functional role of anion channels in cardiac diseases
-
Duan D.Y., Liu L.L., Bozeat N., Huang Z.M., Xiang S.Y., Wang G.L., et al. Functional role of anion channels in cardiac diseases. Acta Pharmacol Sin 2005, 26:265-278.
-
(2005)
Acta Pharmacol Sin
, vol.26
, pp. 265-278
-
-
Duan, D.Y.1
Liu, L.L.2
Bozeat, N.3
Huang, Z.M.4
Xiang, S.Y.5
Wang, G.L.6
-
18
-
-
64249148329
-
Sticky fingers: CaMKII finds a home on another ion channel
-
Anderson M.E. Sticky fingers: CaMKII finds a home on another ion channel. Circ Res 2009, 104:712-714.
-
(2009)
Circ Res
, vol.104
, pp. 712-714
-
-
Anderson, M.E.1
-
20
-
-
77950419553
-
CaV1.2 beta-subunit coordinates CaMKII-triggered cardiomyocyte death and afterdepolarizations
-
Koval O.M., Guan X., Wu Y., Joiner M.L., Gao Z., Chen B., et al. CaV1.2 beta-subunit coordinates CaMKII-triggered cardiomyocyte death and afterdepolarizations. Proc Natl Acad Sci U S A 2010, 107:4996-5000.
-
(2010)
Proc Natl Acad Sci U S A
, vol.107
, pp. 4996-5000
-
-
Koval, O.M.1
Guan, X.2
Wu, Y.3
Joiner, M.L.4
Gao, Z.5
Chen, B.6
-
21
-
-
77957838101
-
A betaIV spectrin/CaMKII signaling complex is essential for membrane excitability in mice
-
Hund T.J., Koval O.M., Li J., Wright P.J., Qian L., Snyder J.S., et al. A betaIV spectrin/CaMKII signaling complex is essential for membrane excitability in mice. J Clin Invest 2010, 120:3508-3519.
-
(2010)
J Clin Invest
, vol.120
, pp. 3508-3519
-
-
Hund, T.J.1
Koval, O.M.2
Li, J.3
Wright, P.J.4
Qian, L.5
Snyder, J.S.6
-
22
-
-
84922392783
-
Ankyrin-G coordinates intercalated disc signaling platform to regulate cardiac excitability in vivo
-
Makara M.A., Curran J., Little S., Musa H., Polina I., Smith S.A., et al. Ankyrin-G coordinates intercalated disc signaling platform to regulate cardiac excitability in vivo. Circ Res 2014, 115:929-938.
-
(2014)
Circ Res
, vol.115
, pp. 929-938
-
-
Makara, M.A.1
Curran, J.2
Little, S.3
Musa, H.4
Polina, I.5
Smith, S.A.6
-
23
-
-
64249158777
-
Kv4 potassium channels form a tripartite complex with the anchoring protein SAP97 and CaMKII in cardiac myocytes
-
El-Haou S., Balse E., Neyroud N., Dilanian G., Gavillet B., Abriel H., et al. Kv4 potassium channels form a tripartite complex with the anchoring protein SAP97 and CaMKII in cardiac myocytes. Circ Res 2009, 104:758-769.
-
(2009)
Circ Res
, vol.104
, pp. 758-769
-
-
El-Haou, S.1
Balse, E.2
Neyroud, N.3
Dilanian, G.4
Gavillet, B.5
Abriel, H.6
-
24
-
-
0038643573
-
The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure
-
Zhang T., Maier L.S., Dalton N.D., Miyamoto S., Ross J., Bers D.M., et al. The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure. Circ Res 2003, 92:912-919.
-
(2003)
Circ Res
, vol.92
, pp. 912-919
-
-
Zhang, T.1
Maier, L.S.2
Dalton, N.D.3
Miyamoto, S.4
Ross, J.5
Bers, D.M.6
-
25
-
-
60549085044
-
The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload
-
Backs J., Backs T., Neef S., Kreusser M.M., Lehmann L.H., Patrick D.M., et al. The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload. Proc Natl Acad Sci U S A 2009, 106:2342-2347.
-
(2009)
Proc Natl Acad Sci U S A
, vol.106
, pp. 2342-2347
-
-
Backs, J.1
Backs, T.2
Neef, S.3
Kreusser, M.M.4
Lehmann, L.H.5
Patrick, D.M.6
-
26
-
-
66449097490
-
2+/calmodulin-dependent kinase II in the transition from pressure overload- induced cardiac hypertrophy to heart failure in mice
-
2+/calmodulin-dependent kinase II in the transition from pressure overload- induced cardiac hypertrophy to heart failure in mice. J Clin Invest 2009, 119:1230-1240.
-
(2009)
J Clin Invest
, vol.119
, pp. 1230-1240
-
-
Ling, H.1
Zhang, T.2
Pereira, L.3
Means, C.K.4
Cheng, H.5
Gu, Y.6
-
27
-
-
84897952396
-
CaMKII in sinoatrial node physiology and dysfunction
-
Wu Y., Anderson M.E. CaMKII in sinoatrial node physiology and dysfunction. Front Pharmacol 2014, 5:48.
-
(2014)
Front Pharmacol
, vol.5
, pp. 48
-
-
Wu, Y.1
Anderson, M.E.2
-
28
-
-
84905050495
-
Arrhythmias, elicited by catecholamines and serotonin, vanish in human chronic atrial fibrillation
-
Christ T., Rozmaritsa N., Engel A., Berk E., Knaut M., Metzner K., et al. Arrhythmias, elicited by catecholamines and serotonin, vanish in human chronic atrial fibrillation. Proc Natl Acad Sci U S A 2014, 111:11193-11198.
-
(2014)
Proc Natl Acad Sci U S A
, vol.111
, pp. 11193-11198
-
-
Christ, T.1
Rozmaritsa, N.2
Engel, A.3
Berk, E.4
Knaut, M.5
Metzner, K.6
-
29
-
-
84901014984
-
Numerical Modeling Calcium and CaMKII Effects in the SA Node
-
Yaniv Y., Maltsev V.A. Numerical Modeling Calcium and CaMKII Effects in the SA Node. Front Pharmacol 2014, 5:58.
-
(2014)
Front Pharmacol
, vol.5
, pp. 58
-
-
Yaniv, Y.1
Maltsev, V.A.2
-
30
-
-
84902346655
-
Metabolic memory of ss-cells controls insulin secretion and is mediated by CaMKII
-
Santos G.J., Ferreira S.M., Ortis F., Rezende L.F., Li C., Naji A., et al. Metabolic memory of ss-cells controls insulin secretion and is mediated by CaMKII. Mol Metab 2014, 3:484-489.
-
(2014)
Mol Metab
, vol.3
, pp. 484-489
-
-
Santos, G.J.1
Ferreira, S.M.2
Ortis, F.3
Rezende, L.F.4
Li, C.5
Naji, A.6
-
31
-
-
84889674935
-
Activation of calcium/calmodulin-dependent protein kinase II in obesity mediates suppression of hepatic insulin signaling
-
Ozcan L., Cristina de Souza J., Harari A.A., Backs J., Olson E.N., Tabas I. Activation of calcium/calmodulin-dependent protein kinase II in obesity mediates suppression of hepatic insulin signaling. Cell Metab 2013, 18:803-815.
-
(2013)
Cell Metab
, vol.18
, pp. 803-815
-
-
Ozcan, L.1
Cristina de Souza, J.2
Harari, A.A.3
Backs, J.4
Olson, E.N.5
Tabas, I.6
-
32
-
-
84901377446
-
High-fat diet-induced mitochondrial biogenesis is regulated by mitochondrial-derived reactive oxygen species activation of CaMKII
-
Jain S.S., Paglialunga S., Vigna C., Ludzki A., Herbst E.A., Lally J.S., et al. High-fat diet-induced mitochondrial biogenesis is regulated by mitochondrial-derived reactive oxygen species activation of CaMKII. Diabetes 2014, 63:1907-1913.
-
(2014)
Diabetes
, vol.63
, pp. 1907-1913
-
-
Jain, S.S.1
Paglialunga, S.2
Vigna, C.3
Ludzki, A.4
Herbst, E.A.5
Lally, J.S.6
-
33
-
-
84870440232
-
CaMKII inhibition rescues pro-arrhythmic phenotypes in model of human ankyrin-B syndrome
-
DeGrande S., Nixon D., Koval O., Curran J.W., Wright P., Wang Q., et al. CaMKII inhibition rescues pro-arrhythmic phenotypes in model of human ankyrin-B syndrome. Heart Rhythm 2012, 9:2034-2041.
-
(2012)
Heart Rhythm
, vol.9
, pp. 2034-2041
-
-
DeGrande, S.1
Nixon, D.2
Koval, O.3
Curran, J.W.4
Wright, P.5
Wang, Q.6
-
36
-
-
57749181755
-
Proarrhythmic defects in Timothy syndrome require calmodulin kinase II
-
Thiel W.H., Chen B., Hund T.J., Koval O.M., Purohit A., Song L.S., et al. Proarrhythmic defects in Timothy syndrome require calmodulin kinase II. Circulation 2008, 118:2225-2234.
-
(2008)
Circulation
, vol.118
, pp. 2225-2234
-
-
Thiel, W.H.1
Chen, B.2
Hund, T.J.3
Koval, O.M.4
Purohit, A.5
Song, L.S.6
-
37
-
-
84895517603
-
CaMKII inhibitors: from research tools to therapeutic agents
-
Pellicena P., Schulman H. CaMKII inhibitors: from research tools to therapeutic agents. Front Pharmacol 2014, 5:21.
-
(2014)
Front Pharmacol
, vol.5
, pp. 21
-
-
Pellicena, P.1
Schulman, H.2
-
38
-
-
84880328248
-
Role of late sodium current as a potential arrhythmogenic mechanism in the progression of pressure-induced heart disease
-
Toischer K., Hartmann N., Wagner S., Fischer T.H., Herting J., Danner B.C., et al. Role of late sodium current as a potential arrhythmogenic mechanism in the progression of pressure-induced heart disease. J Mol Cell Cardiol 2013, 61:111-122.
-
(2013)
J Mol Cell Cardiol
, vol.61
, pp. 111-122
-
-
Toischer, K.1
Hartmann, N.2
Wagner, S.3
Fischer, T.H.4
Herting, J.5
Danner, B.C.6
-
39
-
-
0032898049
-
Repolarization abnormalities in cardiomyocytes of dogs with chronic heart failure: role of sustained inward current
-
Undrovinas A.I., Maltsev V.A., Sabbah H.N. Repolarization abnormalities in cardiomyocytes of dogs with chronic heart failure: role of sustained inward current. Cell Mol Life Sci 1999, 55:494-505.
-
(1999)
Cell Mol Life Sci
, vol.55
, pp. 494-505
-
-
Undrovinas, A.I.1
Maltsev, V.A.2
Sabbah, H.N.3
-
40
-
-
0032497917
-
Ultraslow inactivating sodium current in human ventricular cardiomyocytes
-
Maltsev V.A., Sabbah H.N., Higgins R.S., Silverman N., Lesch M., Novel Undrovinas AI. ultraslow inactivating sodium current in human ventricular cardiomyocytes. Circulation 1998, 98:2545-2552.
-
(1998)
Circulation
, vol.98
, pp. 2545-2552
-
-
Maltsev, V.A.1
Sabbah, H.N.2
Higgins, R.S.3
Silverman, N.4
Lesch, M.5
Novel, U.A.I.6
-
43
-
-
84870954315
-
Mass spectrometry-based identification of native cardiac Nav1.5 channel alpha subunit phosphorylation sites
-
Marionneau C., Lichti C.F., Lindenbaum P., Charpentier F., Nerbonne J.M., Townsend R.R., et al. Mass spectrometry-based identification of native cardiac Nav1.5 channel alpha subunit phosphorylation sites. J Proteome Res 2012, 11:5994-6007.
-
(2012)
J Proteome Res
, vol.11
, pp. 5994-6007
-
-
Marionneau, C.1
Lichti, C.F.2
Lindenbaum, P.3
Charpentier, F.4
Nerbonne, J.M.5
Townsend, R.R.6
-
44
-
-
84903906581
-
The role of late INa in development of cardiac arrhythmias
-
Antzelevitch C., Nesterenko V., Shryock J.C., Rajamani S., Song Y., Belardinelli L. The role of late INa in development of cardiac arrhythmias. Handb Exp Pharmacol 2014, 221:137-168.
-
(2014)
Handb Exp Pharmacol
, vol.221
, pp. 137-168
-
-
Antzelevitch, C.1
Nesterenko, V.2
Shryock, J.C.3
Rajamani, S.4
Song, Y.5
Belardinelli, L.6
-
45
-
-
82455175177
-
Subcellular heterogeneity of sodium current properties in adult cardiac ventricular myocytes
-
Lin X., Liu N., Lu J., Zhang J., Anumonwo J.M., Isom L.L., et al. Subcellular heterogeneity of sodium current properties in adult cardiac ventricular myocytes. Heart Rhythm 2011, 8:1923-1930.
-
(2011)
Heart Rhythm
, vol.8
, pp. 1923-1930
-
-
Lin, X.1
Liu, N.2
Lu, J.3
Zhang, J.4
Anumonwo, J.M.5
Isom, L.L.6
-
46
-
-
15744405775
-
Nav1.5 E1053K mutation causing Brugada syndrome blocks binding to ankyrin-G and expression of Nav1.5 on the surface of cardiomyocytes
-
Mohler P.J., Rivolta I., Napolitano C., LeMaillet G., Lambert S., Priori S.G., et al. Nav1.5 E1053K mutation causing Brugada syndrome blocks binding to ankyrin-G and expression of Nav1.5 on the surface of cardiomyocytes. Proc Natl Acad Sci U S A 2004, 101:17533-17538.
-
(2004)
Proc Natl Acad Sci U S A
, vol.101
, pp. 17533-17538
-
-
Mohler, P.J.1
Rivolta, I.2
Napolitano, C.3
LeMaillet, G.4
Lambert, S.5
Priori, S.G.6
-
47
-
-
33747425933
-
Cardiac sodium channel Nav1.5 is regulated by a multiprotein complex composed of syntrophins and dystrophin
-
Gavillet B., Rougier J.S., Domenighetti A.A., Behar R., Boixel C., Ruchat P., et al. Cardiac sodium channel Nav1.5 is regulated by a multiprotein complex composed of syntrophins and dystrophin. Circ Res 2006, 99:407-414.
-
(2006)
Circ Res
, vol.99
, pp. 407-414
-
-
Gavillet, B.1
Rougier, J.S.2
Domenighetti, A.A.3
Behar, R.4
Boixel, C.5
Ruchat, P.6
-
48
-
-
84904053841
-
PDZ domain-binding motif regulates cardiomyocyte compartment-specific NaV1.5 channel expression and function
-
Shy D., Gillet L., Ogrodnik J., Albesa M., Verkerk A.O., Wolswinkel R., et al. PDZ domain-binding motif regulates cardiomyocyte compartment-specific NaV1.5 channel expression and function. Circulation 2014, 130:147-160.
-
(2014)
Circulation
, vol.130
, pp. 147-160
-
-
Shy, D.1
Gillet, L.2
Ogrodnik, J.3
Albesa, M.4
Verkerk, A.O.5
Wolswinkel, R.6
-
49
-
-
48249148221
-
Syntrophin mutation associated with long QT syndrome through activation of the nNOS-SCN5A macromolecular complex
-
Ueda K., Valdivia C., Medeiros-Domingo A., Tester D.J., Vatta M., Farrugia G., et al. Syntrophin mutation associated with long QT syndrome through activation of the nNOS-SCN5A macromolecular complex. Proc Natl Acad Sci U S A 2008, 105:9355-9360.
-
(2008)
Proc Natl Acad Sci U S A
, vol.105
, pp. 9355-9360
-
-
Ueda, K.1
Valdivia, C.2
Medeiros-Domingo, A.3
Tester, D.J.4
Vatta, M.5
Farrugia, G.6
-
50
-
-
84901034764
-
Modeling CaMKII-mediated regulation of l-type Ca(2+) channels and ryanodine receptors in the heart
-
Greenstein J.L., Foteinou P.T., Hashambhoy-Ramsay Y.L., Winslow R.L. Modeling CaMKII-mediated regulation of l-type Ca(2+) channels and ryanodine receptors in the heart. Front Pharmacol 2014, 5:60.
-
(2014)
Front Pharmacol
, vol.5
, pp. 60
-
-
Greenstein, J.L.1
Foteinou, P.T.2
Hashambhoy-Ramsay, Y.L.3
Winslow, R.L.4
-
51
-
-
84895560919
-
Modeling CaMKII in cardiac physiology: from molecule to tissue
-
Onal B., Unudurthi S.D., Hund T.J. Modeling CaMKII in cardiac physiology: from molecule to tissue. Front Pharmacol 2014, 5:9.
-
(2014)
Front Pharmacol
, vol.5
, pp. 9
-
-
Onal, B.1
Unudurthi, S.D.2
Hund, T.J.3
-
52
-
-
8744246332
-
Rate dependence and regulation of action potential and calcium transient in a canine cardiac ventricular cell model
-
Hund T.J., Rudy Y. Rate dependence and regulation of action potential and calcium transient in a canine cardiac ventricular cell model. Circulation 2004, 110:3168-3174.
-
(2004)
Circulation
, vol.110
, pp. 3168-3174
-
-
Hund, T.J.1
Rudy, Y.2
-
54
-
-
74549157674
-
Oxidized calmodulin kinase II regulates conduction following myocardial infarction: a computational analysis
-
Christensen M.D., Dun W., Boyden P.A., Anderson M.E., Mohler P.J., Hund T.J. Oxidized calmodulin kinase II regulates conduction following myocardial infarction: a computational analysis. PLoS Comput Biol 2009, 5:e1000583.
-
(2009)
PLoS Comput Biol
, vol.5
, pp. e1000583
-
-
Christensen, M.D.1
Dun, W.2
Boyden, P.A.3
Anderson, M.E.4
Mohler, P.J.5
Hund, T.J.6
-
55
-
-
50449107767
-
Role of activated CaMKII in abnormal calcium homeostasis and INa remodeling after myocardial infarction: insights from mathematical modeling
-
Hund T.J., Decker K.F., Kanter E., Mohler P.J., Boyden P.A., Schuessler R.B., et al. Role of activated CaMKII in abnormal calcium homeostasis and INa remodeling after myocardial infarction: insights from mathematical modeling. J Mol Cell Cardiol 2008, 45:420-428.
-
(2008)
J Mol Cell Cardiol
, vol.45
, pp. 420-428
-
-
Hund, T.J.1
Decker, K.F.2
Kanter, E.3
Mohler, P.J.4
Boyden, P.A.5
Schuessler, R.B.6
-
57
-
-
70349435474
-
Regulation of excitation-contraction coupling in mouse cardiac myocytes: integrative analysis with mathematical modelling
-
Koivumaki J.T., Korhonen T., Takalo J., Weckstrom M., Tavi P. Regulation of excitation-contraction coupling in mouse cardiac myocytes: integrative analysis with mathematical modelling. BMC Physiol 2009, 9:16.
-
(2009)
BMC Physiol
, vol.9
, pp. 16
-
-
Koivumaki, J.T.1
Korhonen, T.2
Takalo, J.3
Weckstrom, M.4
Tavi, P.5
-
60
-
-
84874607556
-
Diabetes increases mortality after myocardial infarction by oxidizing CaMKII
-
Luo M., Guan X., Di L., Kutschke W., Gao Z., Yang J., et al. Diabetes increases mortality after myocardial infarction by oxidizing CaMKII. J Clin Invest 2013, 123:1262-1274.
-
(2013)
J Clin Invest
, vol.123
, pp. 1262-1274
-
-
Luo, M.1
Guan, X.2
Di, L.3
Kutschke, W.4
Gao, Z.5
Yang, J.6
-
61
-
-
79961015131
-
Oxidized CaMKII causes sinus node dysfunction in mice
-
Swaminathan P.D., Purohit A., Soni S., Voigt N., Singh M.V., Glukhov A.V., et al. Oxidized CaMKII causes sinus node dysfunction in mice. J Clin Invest 2011, 121:3277-3288.
-
(2011)
J Clin Invest
, vol.121
, pp. 3277-3288
-
-
Swaminathan, P.D.1
Purohit, A.2
Soni, S.3
Voigt, N.4
Singh, M.V.5
Glukhov, A.V.6
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