Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

Journal of Biological Chemistry

Volumn 289, Issue 50, 2014, Pages 34457-34465

  Ma, Zhiyuan ^a   Vosseller, Keith ^a  

^a DREXEL UNIVERSITY COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMINO ACIDS; CELL PROLIFERATION; METABOLISM; PHYSIOLOGY;

CELLULAR SIGNALING; ENERGY METABOLISM; EPIGENETICS; HYDROXYL GROUPS; N-ACETYLGLUCOSAMINE; NUCLEAR PROTEINS; POST-TRANSLATIONAL MODIFICATIONS; THREONINE RESIDUES;

DISEASES;

5 THIOGLUCOSAMINE; N ACETYLGLUCOSAMINE; O LINKED BETA N ACETYLGLUCOSAMINE; PROTEIN INHIBITOR; UNCLASSIFIED DRUG;

ACYLATION; CANCER CELL; CANCER INHIBITION; CARCINOGENICITY; CELL INVASION; CELL METABOLISM; CELL PROLIFERATION; CELL SURVIVAL; DRUG TARGETING; ENERGY METABOLISM; EPIGENETICS; HUMAN; MALIGNANT NEOPLASTIC DISEASE; METASTASIS; NONHUMAN; PROTEIN SYNTHESIS INHIBITION; SHORT SURVEY; SIGNAL TRANSDUCTION; ANIMAL; CARCINOGENESIS; GENETIC EPIGENESIS; GENETICS; METABOLISM; NEOPLASM; NEOVASCULARIZATION (PATHOLOGY); PATHOLOGY; VASCULARIZATION;

ACETYLGLUCOSAMINE; ANIMALS; CARCINOGENESIS; EPIGENESIS, GENETIC; HUMANS; NEOPLASMS; NEOVASCULARIZATION, PATHOLOGIC; SIGNAL TRANSDUCTION;

EID: 84918523488     PISSN: 00219258     EISSN: 1083351X     Source Type: Journal    
DOI: 10.1074/jbc.R114.577718     Document Type: Short Survey

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