Multiple tissue-specific roles for the O-GlcNAc post-translational modification in the induction of and complications arising from type II diabetes

Journal of Biological Chemistry

Volumn 289, Issue 50, 2014, Pages 34466-34471

  Vaidyanathan, Krithika ^a   Wells, Lance ^a  

^a UNIVERSITY OF GEORGIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GENE EXPRESSION; GLUCOSE; INSULIN; TISSUE;

GLUCOSE TOXICITIES; INSULIN RESISTANCE; METABOLIC REGULATIONS; POST-TRANSLATIONAL MODIFICATIONS; TISSUE SPECIFICS; TYPE II DIABETES; TYPE II DIABETES MELLITUS;

GENE EXPRESSION REGULATION;

INSULIN; N ACETYLGLUCOSAMINE;

ADIPOSE TISSUE; ADVERSE OUTCOME; CHEMICAL MODIFICATION; DIABETIC COMPLICATION; ENERGY METABOLISM; GENE EXPRESSION; GLUCONEOGENESIS; GLUCOSE METABOLISM; GLUCOSE TRANSPORT; HUMAN; LIVER; METABOLIC REGULATION; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; PANCREAS ISLET BETA CELL; PANCREAS ISLET CELL FUNCTION; PROTEIN PROCESSING; SHORT SURVEY; SIGNAL TRANSDUCTION; SKELETAL MUSCLE; ANIMAL; ANTIBODY SPECIFICITY; COMPLICATION; METABOLISM; PATHOLOGY;

ACETYLGLUCOSAMINE; ANIMALS; DIABETES MELLITUS, TYPE 2; HUMANS; INSULIN; ORGAN SPECIFICITY; PROTEIN PROCESSING, POST-TRANSLATIONAL; SIGNAL TRANSDUCTION;

EID: 84918508564     PISSN: 00219258     EISSN: 1083351X     Source Type: Journal    
DOI: 10.1074/jbc.R114.591560     Document Type: Short Survey

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