Activation of hexosamine pathway impairs nitric oxide (NO)-dependent arteriolar dilations by increased protein O-GlcNAcylation

Vascular Pharmacology

Volumn 56, Issue 3-4, 2012, Pages 115-121

  Beleznai, Timea ^a,b   Bagi, Zsolt ^a  

^a UNIVERSITY OF OXFORD   (United Kingdom)

^b UNIVERSITY OF DEBRECEN   (Hungary)

Author keywords

Diabetes mellitus; Glucosamine; Hexosamine pathway; Hyperglycemia; Microvessel; O GlcNAcylation

Indexed keywords

ACETYLCHOLINE; AZASERINE; ENDOTHELIAL NITRIC OXIDE SYNTHASE; GLUCOSAMINE; HEXOSAMINE; HISTAMINE; N ACETYLGLUCOSAMINE; N(G) NITROARGININE METHYL ESTER; NITROPRUSSIDE SODIUM; NORADRENALIN; SEROTONIN;

ACYLATION; ANIMAL EXPERIMENT; ARTERIOLE; ARTERY DIAMETER; ARTERY DILATATION; ARTICLE; CONTROLLED STUDY; ENZYME PHOSPHORYLATION; GLUCOSE INTAKE; MALE; MICROVASCULATURE; NONHUMAN; PRIORITY JOURNAL; PROTEIN MODIFICATION; RAT; SYNTHESIS;

ACETYLGLUCOSAMINE; AMINOACYLATION; ANIMALS; ARTERIOLES; DIABETIC ANGIOPATHIES; ENZYME ACTIVATION; ENZYME INHIBITORS; GLUTAMINE-FRUCTOSE-6-PHOSPHATE TRANSAMINASE (ISOMERIZING); HEXOSAMINES; HYPERGLYCEMIA; MALE; MUSCLE, SKELETAL; NITRIC OXIDE; NITRIC OXIDE SYNTHASE TYPE III; PHOSPHORYLATION; PROTEIN PROCESSING, POST-TRANSLATIONAL; RATS; RATS, WISTAR; VASODILATION; VASODILATOR AGENTS;

EID: 84858155133     PISSN: 15371891     EISSN: 18793649     Source Type: Journal    
DOI: 10.1016/j.vph.2011.11.003     Document Type: Article

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