NFATc3 pathway participates in the process that 15-LO/15-HETE protects pulmonary artery smooth muscle cells against apoptosis during hypoxia

Journal of Receptors and Signal Transduction

Volumn 34, Issue 4, 2014, Pages 270-282

  Ran, Yajuan ^a   Wu, Hong ^b   Wei, Liuping ^a   Yu, Xiufeng ^a   Chen, Jianguo ^a   Li, Shanshan ^a,c   Zhang, Lei ^a   Lou, Jianshi ^b   Zhu, Daling ^a,c  

^a HARBIN MEDICAL UNIVERSITY   (China)

^b TIANJIN MEDICAL UNIVERSITY   (China)

^c Bio pharmaceutical Key Laboratory of Heilongjiang Province   (China)

Author keywords

15 HETE; Apoptosis; Hypoxia; NFATc3; Pulmonary hypertension

Indexed keywords

15 HYDROXYICOSATETRAENOIC ACID; ARACHIDONATE 15 LIPOXYGENASE; CYCLOSPORIN A; LIPOCORTIN 5; NORDIHYDROGUAIARETIC ACID; NUCLEAR FACTOR OF ACTIVATED T CELLS ISOFORM C3; SMALL INTERFERING RNA; TRANSCRIPTION FACTOR NFAT; UNCLASSIFIED DRUG; 15-HYDROXY-5,8,11,13-EICOSATETRAENOIC ACID; CALCIUM; HYDROXYICOSATETRAENOIC ACID;

ADULT; ANIMAL CELL; ANIMAL CELL CULTURE; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CALCIUM CELL LEVEL; CELL SURVIVAL; CELL VIABILITY; CHROMATIN CONDENSATION; CONTROLLED STUDY; DNA FRAGMENTATION; HYPOXIA; MALE; MEMBRANE DEPOLARIZATION; MITOCHONDRIAL MEMBRANE POTENTIAL; MORPHOMETRICS; NICK END LABELING; NONHUMAN; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; PULMONARY ARTERY; PULMONARY HYPERTENSION; RAT; SIGNAL TRANSDUCTION; SMOOTH MUSCLE FIBER; UPREGULATION; ANIMAL; CELL HYPOXIA; CELL PROLIFERATION; DRUG EFFECTS; METABOLISM; MOUSE;

ANIMALS; ANNEXIN A5; APOPTOSIS; ARACHIDONATE 15-LIPOXYGENASE; CALCIUM; CELL HYPOXIA; CELL PROLIFERATION; HYDROXYEICOSATETRAENOIC ACIDS; MASOPROCOL; MEMBRANE POTENTIAL, MITOCHONDRIAL; MICE; MYOCYTES, SMOOTH MUSCLE; NFATC TRANSCRIPTION FACTORS; PULMONARY ARTERY; RATS; SIGNAL TRANSDUCTION;

EID: 84905190969     PISSN: 10799893     EISSN: 15324281     Source Type: Journal    
DOI: 10.3109/10799893.2014.917322     Document Type: Article

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