A Role for BLM in Double-Strand Break Repair Pathway Choice: Prevention of CtIP/Mre11-Mediated Alternative Nonhomologous End-Joining

Cell Reports

Volumn 5, Issue 1, 2013, Pages 21-28

  Grabarz, Anastazja ^b   Guirouilh Barbat, Josée ^a,b   Barascu, Aurélia ^a,b   Pennarun, Gaëlle ^b   Genet, Diane ^b   Rass, Emilie ^b   Germann, Susanne M ^c   Bertrand, Pascale ^b   Hickson, Ian D ^c   Lopez, Bernard S ^a,b  

^a INSTITUT GUSTAVE ROUSSY   (France)

^b INSTITUT DE RADIOPROTECTION ET DE SÛRETÉ NUCLÉAIRE   (France)

^c UNIVERSITY OF COPENHAGEN   (Denmark)

Author keywords

[No Author keywords available]

Indexed keywords

BLOOM SYNDROME HELICASE; MRE11 PROTEIN; NUCLEIC ACID BINDING PROTEIN; PROTEIN CTIP; UNCLASSIFIED DRUG;

ARTICLE; CELL CYCLE; CONTROLLED STUDY; DNA DEGRADATION; DNA END JOINING REPAIR; DOUBLE STRANDED DNA BREAK; FIBROBLAST; GENETIC TRANSFECTION; GENOMIC INSTABILITY; HUMAN; HUMAN CELL; IMMUNOFLUORESCENCE; IONIZING RADIATION; PRIORITY JOURNAL; REGULATORY MECHANISM; SEQUENCE ANALYSIS; WESTERN BLOTTING;

AMINO ACID SEQUENCE; CARRIER PROTEINS; CELL LINE, TUMOR; DNA BREAKS, DOUBLE-STRANDED; DNA END-JOINING REPAIR; DNA-BINDING PROTEINS; HUMANS; NUCLEAR PROTEINS; RECQ HELICASES; TRANSFECTION;

EID: 84885838052     PISSN: None     EISSN: 22111247     Source Type: Journal    
DOI: 10.1016/j.celrep.2013.08.034     Document Type: Article

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