ATR and ATM-dependent movement of BLM helicase during replication stress ensures optimal ATM activation and 53BP1 focus formation

Cell Cycle

Volumn 3, Issue 12, 2004, Pages 1579-1586

  Davalos, Albert R ^a   Kaminker, Patrick ^b   Hansen, Rhonda K ^c   Campisi, Judith ^a,b  

^a LAWRENCE BERKELEY NATIONAL LABORATORY   (United States)

^b BUCK INSTITUTE FOR RESEARCH ON AGING   (United States)

^c XOMA Corporation   (United States)

Author keywords

Cancer; DNA damage response; Phosphorylation; PML nuclear body

Indexed keywords

53 BINDING PROTEIN 1; ATM PROTEIN; ATR PROTEIN; BINDING PROTEIN; BLM HELICASE; HELICASE; PROTEIN P53; UNCLASSIFIED DRUG;

ARTICLE; CELL FRACTIONATION; CONTROLLED STUDY; DNA REPAIR; DNA REPLICATION; DNA STRAND BREAKAGE; ENZYME ACTIVITY; HUMAN; HUMAN CELL; IMMUNOFLUORESCENCE; PROTEIN EXPRESSION; PROTEIN LOCALIZATION;

EID: 17144395509     PISSN: 15384101     EISSN: 15514005     Source Type: Journal    
DOI: 10.4161/cc.3.12.1286     Document Type: Article

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