Long-term treadmill exercise inhibits the progression of Alzheimer's disease-like neuropathology in the hippocampus of APP/PS1 transgenic mice

Behavioural Brain Research

Volumn 256, Issue , 2013, Pages 261-272

  Liu, Hui li ^a   Zhao, Gang ^a   Zhang, He ^a   Shi, Li de ^a  

^a CHINA MEDICAL UNIVERSITY   (China)

Author keywords

Amyloid; Alzheimer's disease; APP PS1 transgenic mice; Glycogen synthase kinase 3; Tau; Treadmill exercise

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; CYCLIN DEPENDENT KINASE 5; GLYCOGEN SYNTHASE KINASE 3; PRESENILIN 1; TAU PROTEIN; APP PROTEIN, HUMAN; CDK5 PROTEIN, MOUSE; MAPT PROTEIN, MOUSE; PSEN1 PROTEIN, HUMAN;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DISEASE COURSE; ENZYME INHIBITION; FEMALE; HIPPOCAMPUS; MOUSE; NEUROPATHOLOGY; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION; THERAPY EFFECT; TRANSGENIC MOUSE; TREADMILL EXERCISE; ANIMAL; C57BL MOUSE; DISEASE MODEL; GENETICS; HUMAN; METABOLISM; MOTOR ACTIVITY; PATHOLOGY; PATHOPHYSIOLOGY; PHOSPHORYLATION; PHYSIOLOGY; PLAQUE, AMYLOID; RANDOMIZATION; TIME;

ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; CYCLIN-DEPENDENT KINASE 5; DISEASE MODELS, ANIMAL; FEMALE; GLYCOGEN SYNTHASE KINASE 3; HIPPOCAMPUS; HUMANS; MICE, INBRED C57BL; MICE, TRANSGENIC; MOTOR ACTIVITY; PHOSPHORYLATION; PHYSICAL CONDITIONING, ANIMAL; PLAQUE, AMYLOID; PRESENILIN-1; RANDOM ALLOCATION; SIGNAL TRANSDUCTION; TAU PROTEINS; TIME FACTORS;

EID: 84883691894     PISSN: 01664328     EISSN: 18727549     Source Type: Journal    
DOI: 10.1016/j.bbr.2013.08.008     Document Type: Article

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