P16Ink4a suppression of lung adenocarcinoma by Bmi-1 in the presence of p38 activation

Journal of Thoracic Oncology

Volumn 6, Issue 3, 2011, Pages 423-431

  Lee, Mi Ok ^a   Lee, Hyeon Jae ^b   Kim, Mi Ae ^c   Kim, Eun Kyung ^c   Lee, Ji Hyun ^c   Heo, Jin Hyung ^d   Lee, Seung Hyuk ^a   Cho, Sang Ho ^d   Fornace Jr , Albert J ^e   Jeong, Hye Cheol ^c   Cha, Hyuk Jin ^a  

^a Department of Biomedical Science ^*  (South Korea)

^b Bundang CHA Hospital   (South Korea)

^c Konkuk University   (South Korea)

^d CHA University   (South Korea)

^e GEORGETOWN UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Bmi 1; Lung adenocarcinoma; Methylation; p16Ink4a; p38 activation

Indexed keywords

BMI1 PROTEIN; CYCLIN DEPENDENT KINASE INHIBITOR 2A; MITOGEN ACTIVATED PROTEIN KINASE P38; PROTEIN P53; SYNAPTOPHYSIN;

ADULT; AGED; ANIMAL CELL; ARTICLE; CANCER CELL; CANCER GROWTH; CANCER INHIBITION; CLINICAL ARTICLE; CONTROLLED STUDY; DNA METHYLATION; FEMALE; HUMAN; HUMAN TISSUE; IMMUNOBLOTTING; IMMUNOHISTOCHEMISTRY; LUNG ADENOCARCINOMA; MALE; MOUSE; NONHUMAN; ONCOGENE H RAS; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN EXPRESSION; PROTEIN FUNCTION; REAL TIME POLYMERASE CHAIN REACTION; VIRAL GENE DELIVERY SYSTEM;

EID: 79951769441     PISSN: 15560864     EISSN: 15561380     Source Type: Journal    
DOI: 10.1097/JTO.0b013e3182018ace     Document Type: Article

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