Tsc2 is not a critical target of Akt during normal Drosophila development

Genes and Development

Volumn 18, Issue 20, 2004, Pages 2479-2484

  Dong, Jixin ^a   Pan, Duojia ^a  

^a UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

Author keywords

Cell growth; Insulin signaling; Target of rapamycin (TOR); Tumor suppressor

Indexed keywords

MUTANT PROTEIN; PROTEIN KINASE B; TUBERIN;

ANIMAL CELL; ARTICLE; CELL GROWTH; CONTROLLED STUDY; DROSOPHILA; GENETIC MODEL; INSECT DEVELOPMENT; NONHUMAN; NULL CELL; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; PROTEIN TARGETING;

ANIMALS; ANIMALS, GENETICALLY MODIFIED; CELLS, CULTURED; DNA, COMPLEMENTARY; DROSOPHILA; DROSOPHILA PROTEINS; IMMUNOPRECIPITATION; MODELS, GENETIC; MUTAGENESIS, SITE-DIRECTED; PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-AKT; REPRESSOR PROTEINS; TUMOR SUPPRESSOR PROTEINS; WING;

ANIMALIA; ARACHNIDA; HEXAPODA; INSECTA; TOR;

EID: 5444233787     PISSN: 08909369     EISSN: None     Source Type: Journal    
DOI: 10.1101/gad.1240504     Document Type: Article

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