Mitochondrial Aβ: A potential cause of metabolic dysfunction in Alzheimer's disease

IUBMB Life

Volumn 58, Issue 12, 2006, Pages 686-694

  Chen, Xi ^a   Yan, Shi Du ^b  

^a SAINT LOUIS UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b Columbia University ^*  (United States)

Author keywords

Alzheimer's disease; Amyloid binding alcohol dehydrogenase; Amyloid peptide; Mitochondria

Indexed keywords

ALCOHOL DEHYDROGENASE; AMYLOID BETA PROTEIN; REACTIVE OXYGEN METABOLITE; 3 HYDROXYACYL COENZYME A DEHYDROGENASE; ENZYME; HADH2 PROTEIN, HUMAN; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; BRAIN METABOLISM; BRAIN MITOCHONDRION; CELL MIGRATION; CELL STRESS; CITRIC ACID CYCLE; ENZYME ACTIVITY; ENZYME BINDING; ENZYME DEFECT; ENZYME INHIBITION; ENZYME MECHANISM; GLYCOLYSIS; HUMAN; METABOLIC DISORDER; METABOLITE; MITOCHONDRIAL TOXICITY; MOLECULAR INTERACTION; NONHUMAN; RESPIRATORY CHAIN; REVIEW; SIGNAL TRANSDUCTION; TRANSGENIC MOUSE; ELECTRON TRANSPORT; ENERGY METABOLISM; METABOLISM; MITOCHONDRION; PHYSIOLOGY;

MUS;

3-HYDROXYACYL COA DEHYDROGENASES; ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; ELECTRON TRANSPORT; ENERGY METABOLISM; ENZYMES; HUMANS; MITOCHONDRIA;

EID: 34250211094     PISSN: 15216543     EISSN: 15216551     Source Type: Journal    
DOI: 10.1080/15216540601047767     Document Type: Review

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