Protection of HIF-1-deficient primary renal tubular epithelial cells from hypoxia-induced cell death is glucose dependent

American Journal of Physiology - Renal Physiology

Volumn 289, Issue 6 58-6, 2005, Pages

  Biju, Mangatt P ^a   Akai, Yasuhiro ^a   Shrimanker, Nikita ^a   Haase, Volker H ^a  

^a UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

Apoptosis; Cre loxP mediated recombination; Glycolysis and glucose uptake; Renal proximal tubule

Indexed keywords

CRE RECOMBINASE; GLUCOSE; HYPOXIA INDUCIBLE FACTOR 1; TETRACYCLINE;

ACUTE KIDNEY FAILURE; ADAPTATION; ANGIOGENESIS; ANIMAL CELL; APOPTOSIS; ARTICLE; CELL DEATH; CELL PROLIFERATION; CELL SURVIVAL; CONTROLLED STUDY; DISEASE MODEL; ERYTHROPOIESIS; GENETIC MODEL; GLUCOSE TRANSPORT; GLUCOSE UTILIZATION; GLYCOLYSIS; HYPOXIA; KIDNEY ISCHEMIA; KIDNEY TUBULE EPITHELIUM; LOXP SITE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; TRANSCRIPTION REGULATION;

EID: 27944451518     PISSN: 1931857X     EISSN: 15221466     Source Type: Journal    
DOI: 10.1152/ajprenal.00233.2005     Document Type: Article

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