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Oncogene

Volumn 21-4, Issue 1, 2002, Pages 592-597

Telomere dysfunction: Multiple paths to the same end

(2) Harrington, Lea a Robinson, Murray b

a UNIVERSITY OF TORONTO (Canada)

b AMGEN INC (United States)

Author keywords

Apoptosis; DNA damage; Genome stability; P53; Telomerase; Telomere

Indexed keywords

DNA; DNA BINDING PROTEIN; PROTEIN P53; TELOMERASE; TELOMERIC REPEAT BINDING FACTOR 2; UNCLASSIFIED DRUG;

ANIMAL CELL; APOPTOSIS; CHROMOSOME BREAKAGE; DNA DAMAGE; ENZYME ANALYSIS; GENE DISRUPTION; GENETIC STABILITY; GENOTOXICITY; HUMAN; HUMAN CELL; MAMMAL CELL; MOLECULAR CLONING; MOLECULAR RECOGNITION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROCESS MODEL; PROTEIN ANALYSIS; PROTEIN DEFECT; REVIEW; TELOMERE;

ANIMALS; APOPTOSIS; CELL TRANSFORMATION, NEOPLASTIC; DNA DAMAGE; HUMANS; MICE; MODELS, GENETIC; RNA; SIGNAL TRANSDUCTION; TELOMERASE; TELOMERE; TUMOR SUPPRESSOR PROTEIN P53;

ANIMALIA; MAMMALIA; MURINAE;

EID: 0037148278 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj/onc/1205084 Document Type: Review

Times cited : (58)

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