메뉴 건너뛰기




Volumn 11, Issue 1, 2001, Pages 98-103

Cellular senescence as a tumor-protection mechanism: The essential role of counting

Author keywords

[No Author keywords available]

Indexed keywords

CELL CYCLE; CELL DIVISION; CELL GROWTH; CELL PROLIFERATION; DNA DAMAGE; FIBROBLAST; GENE OVEREXPRESSION; GENE SEQUENCE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; REVIEW; SEQUENCE ANALYSIS; TELOMERE; TUMOR SUPPRESSOR GENE;

EID: 0035253719     PISSN: 0959437X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0959-437X(00)00163-5     Document Type: Review
Times cited : (262)

References (49)
  • 9
    • 0032567967 scopus 로고    scopus 로고
    • Reconstitution of telomerase activity in normal human cells leads to elongation of telomeres and extended replicative life span
    • (1998) Curr Biol , vol.8 , pp. 279-282
    • Vaziri, H.1    Benchimol, S.2
  • 33
  • 39
    • 0033993450 scopus 로고    scopus 로고
    • The p53 tumor suppressor protein does not regulate expression of its own inhibitor, MDM2, except under conditions of stress
    • (2000) Mol Cell Biol , vol.20 , pp. 2023-2030
    • Mendrysa, S.M.1    Perry, M.E.2
  • 41
    • 0033834247 scopus 로고    scopus 로고
    • Telomere dynamics in cancer progression and prevention: Fundamental differences in human and mouse telomere biology
    • (2000) Nat Med , vol.6 , pp. 849-851
    • Wright, W.E.1    Shay, J.W.2
  • 47
    • 0033572894 scopus 로고    scopus 로고
    • Differentiation between senescence (M1) and crisis (M2) in human fibroblast cultures
    • (1999) Exp Cell Res , vol.253 , pp. 519-522
    • Wei, W.1    Sedivy, J.M.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.