Cleavage of Bax is mediated by caspase-dependent or -independent calpain activation in dopaminergic neuronal cells: Protective role of Bcl-2

Journal of Neurochemistry

Volumn 77, Issue 6, 2001, Pages 1531-1541

  Choi, Won Seok ^a   Lee, Eun Hee ^a   Chung, Chul Woong ^b   Jung, Yong Keun ^b   Jin, Byung K ^c   Kim, Seung U ^c   Oh, Tae H ^d   Saido, Takaomi C ^e   Oh, Young J ^a  

^a Yonsei University   (South Korea)

^b Gwangju Institute of Science and Technology (GIST)   (South Korea)

^c Ajou University School of Medicine   (South Korea)

^d UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

^e RIKEN BRAIN SCIENCE INSTITUTE   (Japan)

Author keywords

Bax; Bcl 2; Calpain; Caspase; MPP+; Staurosporine

Indexed keywords

CALPAIN; CASPASE; PROTEIN BAX; PROTEIN BCL 2;

APOPTOSIS; ARTICLE; CELL DEATH; DOPAMINERGIC NERVE CELL; ENZYME ACTIVATION; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROTEIN DEGRADATION;

1-METHYL-4-PHENYLPYRIDINIUM; ANIMALS; BCL-2-ASSOCIATED X PROTEIN; CALPAIN; CASPASES; CELL DEATH; CELL LINE; DOPAMINE; ENZYME INHIBITORS; HERBICIDES; HUMANS; MICROSCOPY, ELECTRON; NECROSIS; NEURONS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; STAUROSPORINE;

UNIDENTIFIED BACULOVIRUS;

EID: 0034967241     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.2001.00368.x     Document Type: Article

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