E2f1 and p53 are dispensable, whereas p21(Waf1/Cip1) cooperates with Rb to restrict endoreduplication and apoptosis during skeletal myogenesis

Developmental Biology

Volumn 227, Issue 1, 2000, Pages 28-41

  Jiang, Zhe ^a   Liang, Ping ^a   Leng, Roger ^a   Guo, Zhong ^a   Liu, Yule ^a   Liu, Xudong ^a   Bubnic, Simon ^a   Keating, Armand ^a   Murray, David ^c   Goss, Paul ^a   Zacksenhaus, Eldad ^a,b  

^a Department of Medicine ^*  (Canada)

^b UNIVERSITY OF TORONTO   (Canada)

^c ST MICHAEL'S HOSPITAL   (Canada)

Author keywords

Apoptosis; Cell cycle; E2F1; Endoreduplication; Myogenesis; P21; P53; Rb

Indexed keywords

PROTEIN P21; PROTEIN P53; RETINOBLASTOMA PROTEIN; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR E2F1; UNCLASSIFIED DRUG; CARRIER PROTEIN; CDKN1A PROTEIN, MOUSE; CELL CYCLE PROTEIN; CYCLIN DEPENDENT KINASE INHIBITOR 1A; CYCLINE; DNA BINDING PROTEIN; E2F1 PROTEIN, MOUSE; MESSENGER RNA; RETINOBLASTOMA BINDING PROTEIN 1; TRANSCRIPTION FACTOR DP1; TRANSCRIPTION FACTOR E2F;

ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL DIFFERENTIATION; DNA SYNTHESIS; EMBRYO; FETUS; GENETIC ANALYSIS; GENOTYPE; MOUSE; MUSCLE DEVELOPMENT; MUTANT; NONHUMAN; PRIORITY JOURNAL; SKELETAL MUSCLE; ANIMAL; BONE DYSPLASIA; CYTOCHEMISTRY; CYTOLOGY; DNA REPLICATION; GENE DELETION; GENETICS; IN SITU HYBRIDIZATION; METABOLISM; MOUSE MUTANT; PATHOLOGY; PHYSIOLOGY; POLYPLOIDY; PRENATAL DEVELOPMENT; SIGNAL TRANSDUCTION;

ANIMALIA;

ANIMALS; APOPTOSIS; BONE DISEASES, DEVELOPMENTAL; CARRIER PROTEINS; CELL CYCLE PROTEINS; CELL DIFFERENTIATION; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; DNA REPLICATION; DNA-BINDING PROTEINS; E2F TRANSCRIPTION FACTORS; E2F1 TRANSCRIPTION FACTOR; EMBRYONIC AND FETAL DEVELOPMENT; GENE DELETION; HISTOCYTOCHEMISTRY; IN SITU HYBRIDIZATION; MICE; MICE, KNOCKOUT; MUSCLE, SKELETAL; POLYPLOIDY; RETINOBLASTOMA PROTEIN; RNA, MESSENGER; SIGNAL TRANSDUCTION; TRANSCRIPTION FACTOR DP1; TRANSCRIPTION FACTORS; TUMOR SUPPRESSOR PROTEIN P53;

EID: 0034331291     PISSN: 00121606     EISSN: None     Source Type: Journal    
DOI: 10.1006/dbio.2000.9892     Document Type: Article

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