Physiological mitochondrial fragmentation is a normal cardiac adaptation to increased energy demand

Circulation Research

Volumn 122, Issue 2, 2018, Pages 282-295

  Coronado, Michael ^a   Fajardo, Giovanni ^a   Nguyen, Kim ^a   Zhao, Mingming ^a   Kooiker, Kristina ^a   Jung, Gwanghyun ^a   Hu, Dong Qing ^a   Reddy, Sushma ^a   Sandoval, Erik ^a   Stotland, Aleksandr ^b   Gottlieb, Roberta A ^b   Bernstein, Daniel ^a  

^a STANFORD UNIVERSITY   (United States)

^b CEDARS SINAI MEDICAL CENTER   (United States)

Author keywords

Cell death; Dynamins; Exercise; Mitochondria; Receptors, adrenergic

Indexed keywords

BETA 1 ADRENERGIC RECEPTOR; DYNAMIN; DYNAMIN RELATED PROTEIN 1; MITOCHONDRIAL DIVISION INHIBITOR 1; PROTEIN INHIBITOR; PROTEIN P110; UNCLASSIFIED DRUG; 3-(2,4-DICHLORO-5-METHOXYPHENYL)-2-SULFANYL-4(3H)-QUINAZOLINONE; QUINAZOLINONE DERIVATIVE;

ADAPTATION; ADULT; ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CELLULAR, SUBCELLULAR AND MOLECULAR BIOLOGICAL PHENOMENA AND FUNCTIONS; CONTROLLED STUDY; DOWN REGULATION; ENERGY CONSUMPTION; EXERCISE; HEART FUNCTION; HEART MITOCHONDRION; MALE; MITOCHONDRIAL FISSION; MITOCHONDRIAL FRAGMENTATION; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOPHAGY; MOUSE; NONHUMAN; PHYSIOLOGICAL STRESS; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; ANIMAL; DRUG EFFECT; ENERGY METABOLISM; KNOCKOUT MOUSE; MITOCHONDRIAL DYNAMICS; PHYSIOLOGY; PROCEDURES;

ADAPTATION, PHYSIOLOGICAL; ANIMALS; ENERGY METABOLISM; MALE; MICE; MICE, KNOCKOUT; MITOCHONDRIAL DYNAMICS; PHYSICAL CONDITIONING, ANIMAL; QUINAZOLINONES;

EID: 85048595869     PISSN: 00097330     EISSN: 15244571     Source Type: Journal    
DOI: 10.1161/CIRCRESAHA.117.310725     Document Type: Article

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