Type 2 diabetes dysregulates glucose metabolism in cardiac progenitor cells

Journal of Biological Chemistry

Volumn 291, Issue 26, 2016, Pages 13634-13648

  Salabei, Joshua K ^a,b   Lorkiewicz, Pawel K ^a,b   Mehra, Parul ^a,b   Gibb, Andrew A ^a,b   Haberzettl, Petra ^a,b   Hong, Kyung U ^a,b   Wei, Xiaoli ^b   Zhang, Xiang ^a,b   Li, Qianhong ^a   Wysoczynski, Marcin ^a,b   Bolli, Roberto ^a,b   Bhatnagar, Aruni ^a,b   Hill, Bradford G ^a,b  

^a University of Louisville School of Medicine   (United States)

^b UNIVERSITY OF LOUISVILLE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CARBON; CELL DEATH; GLUCOSE; ISOTOPES; METABOLISM;

BIOSYNTHETIC PATHWAY; CARBON PARTITIONING; CARDIAC PROGENITORS; GLUCOSE METABOLISM; PENTOSE PHOSPHATES; PHOSPHOFRUCTOKINASE; POTENTIAL MECHANISM; REPARATIVE RESPONSE;

HEART;

6 PHOSPHOFRUCTO 2 KINASE; BETA1 INTEGRIN; CD45 ANTIGEN; CITRATE SYNTHASE; FRUCTOSE 2,6 BISPHOSPHATASE 3; GLYCEROPHOSPHOLIPID; HYDROGEN PEROXIDE; INSULIN; MITOCHONDRIAL DNA; PENTOSE PHOSPHATE; PHOSPHATASE; PROTEIN KINASE B; STEM CELL ANTIGEN 1; THY 1 ANTIGEN; UNCLASSIFIED DRUG; VASCULOTROPIN RECEPTOR 2; GLUCOSE; PFKFB3 PROTEIN, MOUSE;

ACTA2 GENE; ADIPOGENESIS; ANIMAL CELL; ARTICLE; CARBON PARTITIONING; CARDIAC STEM CELL; CELL DEATH; CELL ISOLATION; CELL PROLIFERATION; CELL STRESS; CELL STRUCTURE; CELL SURVIVAL; CONTROLLED STUDY; DISORDERS OF CARBOHYDRATE METABOLISM; ENZYME ACTIVATION; ENZYME ACTIVITY; FLK1 GENE; GENE; GLYCOLYSIS; INSULIN RESISTANCE; LOX GENE; MALE; MOUSE; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; OXYGEN CONSUMPTION; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; TNNT2 GENE; UPREGULATION; ANIMAL; BIOSYNTHESIS; CARDIAC MYOBLAST; EXPERIMENTAL DIABETES MELLITUS; GENE EXPRESSION REGULATION; GENETICS; METABOLISM; PATHOLOGY;

ANIMALS; DIABETES MELLITUS, EXPERIMENTAL; DIABETES MELLITUS, TYPE 2; GENE EXPRESSION REGULATION, ENZYMOLOGIC; GLUCOSE; GLYCOLYSIS; MALE; MICE; MYOBLASTS, CARDIAC; PHOSPHOFRUCTOKINASE-2; UP-REGULATION;

EID: 84976443398     PISSN: 00219258     EISSN: 1083351X     Source Type: Journal    
DOI: 10.1074/jbc.M116.722496     Document Type: Article

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