Polypathology and dementia after brain trauma: Does brain injury trigger distinct neurodegenerative diseases, or should they be classified together as traumatic encephalopathy?

Experimental Neurology

Volumn 275, Issue , 2016, Pages 381-388

  Washington, Patricia M ^a,b   Villapol, Sonia ^c   Burns, Mark P ^c  

^a NewYork Presbyterian Hospital   (United States)

^b Columbia University ^*  (United States)

^c GEORGETOWN UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

Alzheimer's disease (AD); Amyloid (A ); Chronic traumatic encephalopathy (CTE); Dementia; Tau; Tauopathy; Traumatic brain injury (TBI)

Indexed keywords

AMYLOID BETA PROTEIN; APOLIPOPROTEIN E4; TAR DNA BINDING PROTEIN; TAU PROTEIN;

ALZHEIMER DISEASE; AMYLOID PLAQUE; APOE4 GENE; BRAIN DYSFUNCTION; COGNITIVE DEFECT; DEGENERATIVE DISEASE; DEMENTIA; DISEASE ASSOCIATION; DISEASE CLASSIFICATION; DISEASE SEVERITY; GENE; GENOTYPE; HUMAN; LEWY BODY; NEUROPATHOLOGY; NONHUMAN; PARKINSON DISEASE; PATHOGENESIS; POLYPATHOLOGY; PRIORITY JOURNAL; PROTEIN AGGREGATION; PROTEIN CLEAVAGE; REVIEW; SURVIVAL TIME; TRAUMATIC BRAIN INJURY; ANIMAL; BRAIN; BRAIN INJURIES; BRAIN INJURY, CHRONIC; CLASSIFICATION; COMPLICATION; NEURODEGENERATIVE DISEASES; PATHOLOGY;

ALZHEIMER DISEASE; ANIMALS; BRAIN; BRAIN INJURIES; BRAIN INJURY, CHRONIC; DEMENTIA; HUMANS; NEURODEGENERATIVE DISEASES; PLAQUE, AMYLOID;

EID: 84949725618     PISSN: 00144886     EISSN: 10902430     Source Type: Journal    
DOI: 10.1016/j.expneurol.2015.06.015     Document Type: Review

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