The role of primary cilia in corpus callosum formation is mediated by production of the Gli3 repressor

Human Molecular Genetics

Volumn 24, Issue 17, 2015, Pages 4997-5014

  Laclef, Christine ^a,b   Anselme, Isabelle ^a,b   Besse, Laurianne ^a,b   Catala, Martin ^a,b,d   Palmyre, Aurélien ^a,b   Baas, Dominique ^e,f   Paschaki, Marie ^e,f   Pedraza, Maria ^c,g   Métin, Christine ^c,g   Durand, Bénédicte ^e,f   Schneider Maunoury, Sylvie ^a,b  

^a Sorbonne Universités   (France)

^b INSERM   (France)

^c INSTITUT DU FER À MOULIN   (France)

^d PITIÉ SALPÊTRIÈRE HOSPITAL   (France)

^e UNIVERSITÉ LYON 1   (France)

^f CNRS   (France)

^g SORBONNE UNIVERSITÉ   (France)

Author keywords

[No Author keywords available]

Indexed keywords

BRAIN PROTEIN; PROTEIN RPGRIP1L; TRANSCRIPTION FACTOR GLI3; UNCLASSIFIED DRUG; DNA BINDING PROTEIN; FTM PROTEIN, MOUSE; GLI3 PROTEIN, MOUSE; KRUPPEL LIKE FACTOR; NERVE PROTEIN; REGULATORY FACTOR X TRANSCRIPTION FACTORS; SIGNAL TRANSDUCING ADAPTOR PROTEIN; TRANSCRIPTION FACTOR;

ALLELE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; CORPUS CALLOSUM; CORPUS CALLOSUM AGENESIS; EUKARYOTIC FLAGELLUM; FETUS; FOREBRAIN; MORPHOGENESIS; MOUSE; NERVE CELL; NERVE FIBER; NEUROEPITHELIUM CELL; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; TELENCEPHALON; ANIMAL; CILIARY DYSKINESIA; DEFICIENCY; DISEASE MODEL; EMBRYOLOGY; ENCEPHALOCELE; ENZYMOLOGY; GENE EXPRESSION REGULATION; GENETICS; HUMAN; KIDNEY POLYCYSTIC DISEASE; KNOCKOUT MOUSE; METABOLISM; MUTATION; NEOCORTEX; PATHOLOGY;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; AGENESIS OF CORPUS CALLOSUM; ANIMALS; BODY PATTERNING; CILIA; CILIARY MOTILITY DISORDERS; CORPUS CALLOSUM; DISEASE MODELS, ANIMAL; DNA-BINDING PROTEINS; ENCEPHALOCELE; GENE EXPRESSION REGULATION, DEVELOPMENTAL; HUMANS; KRUPPEL-LIKE TRANSCRIPTION FACTORS; MICE; MICE, KNOCKOUT; MUTATION; NEOCORTEX; NERVE TISSUE PROTEINS; NEURONS; POLYCYSTIC KIDNEY DISEASES; TRANSCRIPTION FACTORS;

EID: 84941914150     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/ddv221     Document Type: Article

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