Genetic deficit of KCa3.1 channels protects against pulmonary circulatory collapse induced by TRPV4 channel activation

British Journal of Pharmacology

Volumn 172, Issue 18, 2015, Pages 4493-4505

  Wandall Frostholm, Christine ^a   Dalsgaard, Thomas ^a   Bajoriunas, Vytis ^a   Oliván Viguera, Aida ^c   Sadda, Veeruanjaneyulu ^a   Beck, Lilliana ^a   Mogensen, Susie ^a   Stankevicius, Edgaras ^b   Simonsen, Ulf ^a   Köhler, Ralf ^c  

^a AARHUS UNIVERSITY   (Denmark)

^b Faculty of Medicine   (Lithuania)

^c INSTITUTO ARAGONÉS DE CIENCIAS DE LA SALUD   (Spain)

Author keywords

[No Author keywords available]

Indexed keywords

POTASSIUM CHANNEL; POTASSIUM CHANNEL KCA3.1; UNCLASSIFIED DRUG; VANILLOID RECEPTOR 4;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CHANNEL GATING; CONTROLLED STUDY; ENDOTHELIUM CELL; EXTRAVASATION; FEMALE; HEART ARREST; HEART RIGHT VENTRICLE PRESSURE; HUMAN; HUMAN CELL; IN VITRO STUDY; LUNG BLOOD VESSEL; LUNG HEMORRHAGE; LUNG PRESSURE; MALE; MEMBRANE CURRENT; MOUSE; NONHUMAN; POTASSIUM CURRENT; PRIORITY JOURNAL; PULMONARY ARTERY; SIGNAL TRANSDUCTION; VASODILATATION;

EID: 84940461786     PISSN: 00071188     EISSN: 14765381     Source Type: Journal    
DOI: 10.1111/bph.13234     Document Type: Article

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