-
1
-
-
0030715323
-
Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade
-
Li P, Nijhawan D, Budihardjo I, Srinivasula SM, Ahmad M, Alnemri ES et al. Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade. Cell 1997; 91: 479-489.
-
(1997)
Cell
, vol.91
, pp. 479-489
-
-
Li, P.1
Nijhawan, D.2
Budihardjo, I.3
Srinivasula, S.M.4
Ahmad, M.5
Alnemri, E.S.6
-
2
-
-
43049152912
-
TNF-alpha induces two distinct caspase-8 activation pathways
-
Wang L, Du F, Wang X. TNF-alpha induces two distinct caspase-8 activation pathways. Cell 2008; 133: 693-703.
-
(2008)
Cell
, vol.133
, pp. 693-703
-
-
Wang, L.1
Du, F.2
Wang, X.3
-
3
-
-
33644840693
-
Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury
-
Degterev A, Huang Z, Boyce M, Li Y, Jagtap P, Mizushima N et al. Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury. Nat Chem Biol 2005; 1: 112-119.
-
(2005)
Nat Chem Biol
, vol.1
, pp. 112-119
-
-
Degterev, A.1
Huang, Z.2
Boyce, M.3
Li, Y.4
Jagtap, P.5
Mizushima, N.6
-
5
-
-
5944233768
-
Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule
-
Holler N, Zaru R, Micheau O, Thome M, Attinger A, Valitutti S et al. Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule. Nat Immunol 2000; 1: 489-495.
-
(2000)
Nat Immunol
, vol.1
, pp. 489-495
-
-
Holler, N.1
Zaru, R.2
Micheau, O.3
Thome, M.4
Attinger, A.5
Valitutti, S.6
-
6
-
-
0029760732
-
RIP and FADD: Two"death domain"-containing proteins can induce apoptosis by convergent, but dissociable, pathways
-
Grimm S, Stanger BZ, Leder P. RIP and FADD: two "death domain"-containing proteins can induce apoptosis by convergent, but dissociable, pathways. Proc Natl Acad Sci USA 1996; 93: 10923-10927.
-
(1996)
Proc Natl Acad Sci USA
, vol.93
, pp. 10923-10927
-
-
Grimm, S.1
Stanger, B.Z.2
Leder, P.3
-
7
-
-
0041853690
-
Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes
-
Micheau O, Tschopp J. Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes. Cell 2003; 114: 181-190.
-
(2003)
Cell
, vol.114
, pp. 181-190
-
-
Micheau, O.1
Tschopp, J.2
-
8
-
-
0033546276
-
RIP3, a novel apoptosisinducing kinase
-
Sun X, Lee J, Navas T, Baldwin DT, Stewart TA, Dixit VM. RIP3, a novel apoptosisinducing kinase. J Biol Chem 1999; 274: 16871-16875.
-
(1999)
J Biol Chem
, vol.274
, pp. 16871-16875
-
-
Sun, X.1
Lee, J.2
Navas, T.3
Baldwin, D.T.4
Stewart, T.A.5
Dixit, V.M.6
-
9
-
-
0033587070
-
Identification of RIP3, a RIP-like kinase that activates apoptosis and NFkappaB
-
Yu PW, Huang BC, Shen M, Quast J, Chan E, Xu X et al. Identification of RIP3, a RIP-like kinase that activates apoptosis and NFkappaB. Curr Biol 1999; 9: 539-542.
-
(1999)
Curr Biol
, vol.9
, pp. 539-542
-
-
Yu, P.W.1
Huang, B.C.2
Shen, M.3
Quast, J.4
Chan, E.5
Xu, X.6
-
10
-
-
0034685982
-
The RIP-like kinase, RIP3, induces apoptosis and NF-kappaB nuclear translocation and localizes to mitochondria
-
Kasof GM, Prosser JC, Liu D, Lorenzi MV, Gomes BC. The RIP-like kinase, RIP3, induces apoptosis and NF-kappaB nuclear translocation and localizes to mitochondria. FEBS Lett 2000; 473: 285-291.
-
(2000)
FEBS Lett
, vol.473
, pp. 285-291
-
-
Kasof, G.M.1
Prosser, J.C.2
Liu, D.3
Lorenzi, M.V.4
Gomes, B.C.5
-
11
-
-
0032862986
-
Mouse receptor interacting protein 3 does not contain a caspase-recruiting or a death domain but induces apoptosis and activates NF-kappaB
-
Pazdernik NJ, Donner DB, Goebl MG, Harrington MA. Mouse receptor interacting protein 3 does not contain a caspase-recruiting or a death domain but induces apoptosis and activates NF-kappaB. Mol Cell Biol 1999; 19: 6500-6508.
-
(1999)
Mol Cell Biol
, vol.19
, pp. 6500-6508
-
-
Pazdernik, N.J.1
Donner, D.B.2
Goebl, M.G.3
Harrington, M.A.4
-
12
-
-
66749183275
-
Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha
-
He S, Wang L, Miao L, Wang T, Du F, Zhao L et al. Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha. Cell 2009; 137: 1100-1111.
-
(2009)
Cell
, vol.137
, pp. 1100-1111
-
-
He, S.1
Wang, L.2
Miao, L.3
Wang, T.4
Du, F.5
Zhao, L.6
-
13
-
-
79952811804
-
RIP3 mediates the embryonic lethality of caspase-8-deficient mice
-
Kaiser WJ, Upton JW, Long AB, Livingston-Rosanoff D, Daley-Bauer LP, Hakem R et al. RIP3 mediates the embryonic lethality of caspase-8-deficient mice. Nature 2011; 471: 368-372.
-
(2011)
Nature
, vol.471
, pp. 368-372
-
-
Kaiser, W.J.1
Upton, J.W.2
Long, A.B.3
Livingston-Rosanoff, D.4
Daley-Bauer, L.P.5
Hakem, R.6
-
14
-
-
84255162797
-
RIP kinase-dependent necrosis drives lethal systemic inflammatory response syndrome
-
Duprez L, Takahashi N, Van Hauwermeiren F, Vandendriessche B, Goossens V, Vanden Berghe T et al. RIP kinase-dependent necrosis drives lethal systemic inflammatory response syndrome. Immunity 2011; 35: 908-918.
-
(2011)
Immunity
, vol.35
, pp. 908-918
-
-
Duprez, L.1
Takahashi, N.2
Van Hauwermeiren, F.3
Vandendriessche, B.4
Goossens, V.5
Vanden Berghe, T.6
-
15
-
-
84862907788
-
Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase
-
Sun L, Wang H, Wang Z, He S, Chen S, Liao D et al. Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase. Cell 2012; 148: 213-227.
-
(2012)
Cell
, vol.148
, pp. 213-227
-
-
Sun, L.1
Wang, H.2
Wang, Z.3
He, S.4
Chen, S.5
Liao, D.6
-
16
-
-
84884308522
-
The pseudokinase MLKL mediates necroptosis via a molecular switch mechanism
-
Murphy JM, Czabotar PE, Hildebrand JM, Lucet IS, Zhang JG, Alvarez-Diaz S et al. The pseudokinase MLKL mediates necroptosis via a molecular switch mechanism. Immunity 2013; 39: 443-453.
-
(2013)
Immunity
, vol.39
, pp. 443-453
-
-
Murphy, J.M.1
Czabotar, P.E.2
Hildebrand, J.M.3
Lucet, I.S.4
Zhang, J.G.5
Alvarez-Diaz, S.6
-
17
-
-
84859467770
-
Mixed lineage kinase domain-like is a key receptor interacting protein 3 downstream component of TNF-induced necrosis
-
Zhao J, Jitkaew S, Cai Z, Choksi S, Li Q, Luo J et al. Mixed lineage kinase domain-like is a key receptor interacting protein 3 downstream component of TNF-induced necrosis. Proc Natl Acad Sci USA 2012; 109: 5322-5327.
-
(2012)
Proc Natl Acad Sci USA
, vol.109
, pp. 5322-5327
-
-
Zhao, J.1
Jitkaew, S.2
Cai, Z.3
Choksi, S.4
Li, Q.5
Luo, J.6
-
18
-
-
84891343566
-
Plasma membrane translocation of trimerized MLKL protein is required for TNF-induced necroptosis
-
Cai Z, Jitkaew S, Zhao J, Chiang HC, Choksi S, Liu J et al. Plasma membrane translocation of trimerized MLKL protein is required for TNF-induced necroptosis. Nat Cell Biol 2014; 16: 55-65.
-
(2014)
Nat Cell Biol
, vol.16
, pp. 55-65
-
-
Cai, Z.1
Jitkaew, S.2
Zhao, J.3
Chiang, H.C.4
Choksi, S.5
Liu, J.6
-
19
-
-
84891739370
-
Translocation of mixed lineage kinase domain-like protein to plasma membrane leads to necrotic cell death
-
Chen X, Li W, Ren J, Huang D, He WT, Song Y et al. Translocation of mixed lineage kinase domain-like protein to plasma membrane leads to necrotic cell death. Cell Res 2014; 24: 105-121.
-
(2014)
Cell Res
, vol.24
, pp. 105-121
-
-
Chen, X.1
Li, W.2
Ren, J.3
Huang, D.4
He, W.T.5
Song, Y.6
-
20
-
-
84898027331
-
Mixed lineage kinase domain-like protein MLKL causes necrotic membrane disruption upon phosphorylation by RIP3
-
Wang H, Sun L, Su L, Rizo J, Liu L, Wang LF et al. Mixed lineage kinase domain-like protein MLKL causes necrotic membrane disruption upon phosphorylation by RIP3. Mol Cell 2014; 54: 133-146.
-
(2014)
Mol Cell
, vol.54
, pp. 133-146
-
-
Wang, H.1
Sun, L.2
Su, L.3
Rizo, J.4
Liu, L.5
Wang, L.F.6
-
21
-
-
84859072803
-
Signal transduction by tumor necrosis factor receptors
-
Cabal-Hierro L, Lazo PS. Signal transduction by tumor necrosis factor receptors. Cell Sig 2012; 24: 1297-1305.
-
(2012)
Cell Sig
, vol.24
, pp. 1297-1305
-
-
Cabal-Hierro, L.1
Lazo, P.S.2
-
22
-
-
34548437549
-
Cleavage of RIP3 inactivates its caspase-independent apoptosis pathway by removal of kinase domain
-
Feng S, Yang Y, Mei Y, Ma L, Zhu DE, Hoti N et al. Cleavage of RIP3 inactivates its caspase-independent apoptosis pathway by removal of kinase domain. Cell Sig 2007; 19: 2056-2067.
-
(2007)
Cell Sig
, vol.19
, pp. 2056-2067
-
-
Feng, S.1
Yang, Y.2
Mei, Y.3
Ma, L.4
Zhu, D.E.5
Hoti, N.6
-
23
-
-
67650812332
-
RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis
-
Zhang DW, Shao J, Lin J, Zhang N, Lu BJ, Lin SC et al. RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis. Science 2009; 325: 332-336.
-
(2009)
Science
, vol.325
, pp. 332-336
-
-
Zhang, D.W.1
Shao, J.2
Lin, J.3
Zhang, N.4
Lu, B.J.5
Lin, S.C.6
-
24
-
-
84862121867
-
The flick of a switch: Which death program to choose?
-
Vandenabeele P, Melino G. The flick of a switch: which death program to choose? Cell Death Diff 2012; 19: 1093-1095.
-
(2012)
Cell Death Diff
, vol.19
, pp. 1093-1095
-
-
Vandenabeele, P.1
Melino, G.2
-
25
-
-
84881075820
-
RIP3: A molecular switch for necrosis and inflammation
-
Moriwaki K, Chan FK. RIP3: a molecular switch for necrosis and inflammation. Genes Dev 2013; 27: 1640-1649.
-
(2013)
Genes Dev
, vol.27
, pp. 1640-1649
-
-
Moriwaki, K.1
Chan, F.K.2
-
26
-
-
0029807304
-
Activation of the Raf-1 kinase cascade by coumermycin-induced dimerization
-
Farrar MA, Alberol-Ila J, Perlmutter RM. Activation of the Raf-1 kinase cascade by coumermycin-induced dimerization. Nature 1996; 383: 178-181.
-
(1996)
Nature
, vol.383
, pp. 178-181
-
-
Farrar, M.A.1
Alberol-Ila, J.2
Perlmutter, R.M.3
-
27
-
-
79960921946
-
The Ripoptosome, a signaling platform that assembles in response to genotoxic stress and loss of IAPs
-
Tenev T, Bianchi K, Darding M, Broemer M, Langlais C, Wallberg F et al. The Ripoptosome, a signaling platform that assembles in response to genotoxic stress and loss of IAPs. Mol Cell 2011; 43: 432-448.
-
(2011)
Mol Cell
, vol.43
, pp. 432-448
-
-
Tenev, T.1
Bianchi, K.2
Darding, M.3
Broemer, M.4
Langlais, C.5
Wallberg, F.6
-
28
-
-
79960922705
-
CIAPs block Ripoptosome formation, a RIP1/caspase-8 containing intracellular cell death complex differentially regulated by cFLIP isoforms
-
Feoktistova M, Geserick P, Kellert B, Dimitrova DP, Langlais C, Hupe M et al. cIAPs block Ripoptosome formation, a RIP1/caspase-8 containing intracellular cell death complex differentially regulated by cFLIP isoforms. Mol Cell 2011; 43: 449-463.
-
(2011)
Mol Cell
, vol.43
, pp. 449-463
-
-
Feoktistova, M.1
Geserick, P.2
Kellert, B.3
Dimitrova, D.P.4
Langlais, C.5
Hupe, M.6
-
29
-
-
0032482169
-
Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor
-
Vercammen D, Beyaert R, Denecker G, Goossens V, Van Loo G, Declercq W et al. Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor. J Exp Med 1998; 187: 1477-1485.
-
(1998)
J Exp Med
, vol.187
, pp. 1477-1485
-
-
Vercammen, D.1
Beyaert, R.2
Denecker, G.3
Goossens, V.4
Van Loo, G.5
Declercq, W.6
-
30
-
-
0041666539
-
Q-VD-OPh, a broad spectrum caspase inhibitor with potent antiapoptotic properties
-
Caserta TM, Smith AN, Gultice AD, Reedy MA, Brown TL. Q-VD-OPh, a broad spectrum caspase inhibitor with potent antiapoptotic properties. Apoptosis 2003; 8: 345-352.
-
(2003)
Apoptosis
, vol.8
, pp. 345-352
-
-
Caserta, T.M.1
Smith, A.N.2
Gultice, A.D.3
Reedy, M.A.4
Brown, T.L.5
-
31
-
-
84860381453
-
Intermediate domain of receptor-interacting protein kinase 1 (RIPK1) determines switch between necroptosis and RIPK1 kinase-dependent apoptosis
-
Duprez L, Bertrand MJ, Vanden Berghe T, Dondelinger Y, Festjens N, Vandenabeele P. Intermediate domain of receptor-interacting protein kinase 1 (RIPK1) determines switch between necroptosis and RIPK1 kinase-dependent apoptosis. J Biol Chem 2012; 287: 14863-14872.
-
(2012)
J Biol Chem
, vol.287
, pp. 14863-14872
-
-
Duprez, L.1
Bertrand, M.J.2
Vanden Berghe, T.3
Dondelinger, Y.4
Festjens, N.5
Vandenabeele, P.6
-
33
-
-
84878964122
-
TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1
-
Moujalled DM, Cook WD, Okamoto T, Murphy J, Lawlor KE, Vince JE et al. TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1. Cell Death Dis 2013; 4: e465.
-
(2013)
Cell Death Dis
, vol.4
, pp. e465
-
-
Moujalled, D.M.1
Cook, W.D.2
Okamoto, T.3
Murphy, J.4
Lawlor, K.E.5
Vince, J.E.6
-
34
-
-
66449133280
-
Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation
-
Cho YS, Challa S, Moquin D, Genga R, Ray TD, Guildford M et al. Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation. Cell 2009; 137: 1112-1123.
-
(2009)
Cell
, vol.137
, pp. 1112-1123
-
-
Cho, Y.S.1
Challa, S.2
Moquin, D.3
Genga, R.4
Ray, T.D.5
Guildford, M.6
-
35
-
-
0034648648
-
Activation of death-inducing signaling complex (DISC) by proapoptotic C-terminal fragment of RIP
-
Kim JW, Choi EJ, Joe CO. Activation of death-inducing signaling complex (DISC) by proapoptotic C-terminal fragment of RIP. Oncogene 2000; 19: 4491-4499.
-
(2000)
Oncogene
, vol.19
, pp. 4491-4499
-
-
Kim, J.W.1
Choi, E.J.2
Joe, C.O.3
-
36
-
-
0023885305
-
The protein kinase family: Conserved features and deduced phylogeny of the catalytic domains
-
Hanks SK, Quinn AM, Hunter T. The protein kinase family: conserved features and deduced phylogeny of the catalytic domains. Science 1988; 241: 42-52.
-
(1988)
Science
, vol.241
, pp. 42-52
-
-
Hanks, S.K.1
Quinn, A.M.2
Hunter, T.3
-
37
-
-
57549098807
-
The Catalogue of SomaticMutations in Cancer (COSMIC)
-
Chapter 10, Unit 10.11
-
Forbes SA, Bhamra G, Bamford S, Dawson E, Kok C, Clements J et al. The Catalogue of SomaticMutations in Cancer (COSMIC). Curr Protoc Hum Genet 2008; Chapter 10, Unit 10.11.
-
(2008)
Curr Protoc Hum Genet
-
-
Forbes, S.A.1
Bhamra, G.2
Bamford, S.3
Dawson, E.4
Kok, C.5
Clements, J.6
-
38
-
-
33845197964
-
Surface comparison of active and inactive protein kinases identifies a conserved activation mechanism
-
Kornev AP, Haste NM, Taylor SS, Eyck LF. Surface comparison of active and inactive protein kinases identifies a conserved activation mechanism. Proc Natl Acad Sci USA 2006; 103: 17783-17788.
-
(2006)
Proc Natl Acad Sci USA
, vol.103
, pp. 17783-17788
-
-
Kornev, A.P.1
Haste, N.M.2
Taylor, S.S.3
Eyck, L.F.4
-
39
-
-
0032579490
-
An induced proximity model for caspase-8 activation
-
Muzio M, Stockwell BR, Stennicke HR, Salvesen GS, Dixit VM. An induced proximity model for caspase-8 activation. J Biol Chem 1998; 273: 2926-2930.
-
(1998)
J Biol Chem
, vol.273
, pp. 2926-2930
-
-
Muzio, M.1
Stockwell, B.R.2
Stennicke, H.R.3
Salvesen, G.S.4
Dixit, V.M.5
-
40
-
-
77952784381
-
Inducible dimerization and inducible cleavage reveal a requirement for both processes in caspase-8 activation
-
Oberst A, Pop C, Tremblay AG, Blais V, Denault JB, Salvesen GS et al. Inducible dimerization and inducible cleavage reveal a requirement for both processes in caspase-8 activation. J Biol Chem 2010; 285: 16632-16642.
-
(2010)
J Biol Chem
, vol.285
, pp. 16632-16642
-
-
Oberst, A.1
Pop, C.2
Tremblay, A.G.3
Blais, V.4
Denault, J.B.5
Salvesen, G.S.6
-
41
-
-
0030199152
-
Functional analysis of Fas signaling in vivo using synthetic inducers of dimerization
-
Spencer DM, Belshaw PJ, Chen L, Ho SN, Randazzo F, Crabtree GR et al. Functional analysis of Fas signaling in vivo using synthetic inducers of dimerization. Curr Biol 1996; 6: 839-847.
-
(1996)
Curr Biol
, vol.6
, pp. 839-847
-
-
Spencer, D.M.1
Belshaw, P.J.2
Chen, L.3
Ho, S.N.4
Randazzo, F.5
Crabtree, G.R.6
-
42
-
-
84897088275
-
Activity of protein kinase RIPK3 determines whether cells die by necroptosis or apoptosis
-
Newton K, Dugger DL, Wickliffe KE, Kapoor N, Cristina de-Almagro M, Vucic D et al. Activity of protein kinase RIPK3 determines whether cells die by necroptosis or apoptosis. Science 2014; 343: 1357-1360.
-
(2014)
Science
, vol.343
, pp. 1357-1360
-
-
Newton, K.1
Dugger, D.L.2
Wickliffe, K.E.3
Kapoor, N.4
Cristina De-Almagro, M.5
Vucic, D.6
-
43
-
-
0033214236
-
Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis
-
Lin Y, Devin A, Rodriguez Y, Liu ZG. Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis. Genes Dev 1999; 13: 2514-2526.
-
(1999)
Genes Dev
, vol.13
, pp. 2514-2526
-
-
Lin, Y.1
Devin, A.2
Rodriguez, Y.3
Liu, Z.G.4
-
44
-
-
84856160569
-
Caspase 8 inhibits programmed necrosis by processing CYLD
-
O'Donnell MA, Perez-Jimenez E, Oberst A, Ng A, Massoumi R, Xavier R et al. Caspase 8 inhibits programmed necrosis by processing CYLD. Nat Cell Biol 2011; 13: 1437-1442.
-
(2011)
Nat Cell Biol
, vol.13
, pp. 1437-1442
-
-
O'Donnell, M.A.1
Perez-Jimenez, E.2
Oberst, A.3
Ng, A.4
Massoumi, R.5
Xavier, R.6
-
45
-
-
84888413953
-
Positive and negative phosphorylation regulates RIP1-and RIP3-induced programmed necrosis
-
McQuade T, Cho Y, Chan FK. Positive and negative phosphorylation regulates RIP1-and RIP3-induced programmed necrosis. Biochem J 2013; 456: 409-415.
-
(2013)
Biochem J
, vol.456
, pp. 409-415
-
-
McQuade, T.1
Cho, Y.2
Chan, F.K.3
-
46
-
-
36148954336
-
IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis
-
Vince JE, Wong WW, Khan N, Feltham R, Chau D, Ahmed AU et al. IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis. Cell 2007; 131: 682-693.
-
(2007)
Cell
, vol.131
, pp. 682-693
-
-
Vince, J.E.1
Wong, W.W.2
Khan, N.3
Feltham, R.4
Chau, D.5
Ahmed, A.U.6
-
47
-
-
84886095480
-
Lymphotoxin alpha induces apoptosis, necroptosis and inflammatory signals with the same potency as tumour necrosis factor
-
Etemadi N, Holien JK, Chau D, Dewson G, Murphy JM, Alexander WS et al. Lymphotoxin alpha induces apoptosis, necroptosis and inflammatory signals with the same potency as tumour necrosis factor. FEBS J 2013; 280: 5283-5297.
-
(2013)
FEBS J
, vol.280
, pp. 5283-5297
-
-
Etemadi, N.1
Holien, J.K.2
Chau, D.3
Dewson, G.4
Murphy, J.M.5
Alexander, W.S.6
-
48
-
-
84890921845
-
A robust methodology to subclassify pseudokinases based on their nucleotide-binding properties
-
Murphy JM, Zhang Q, Young SN, Reese ML, Bailey FP, Eyers PA et al. A robust methodology to subclassify pseudokinases based on their nucleotide-binding properties. Biochem J 2014; 457: 323-334.
-
(2014)
Biochem J
, vol.457
, pp. 323-334
-
-
Murphy, J.M.1
Zhang, Q.2
Young, S.N.3
Reese, M.L.4
Bailey, F.P.5
Eyers, P.A.6
-
49
-
-
84878765817
-
In vitro JAK kinase activity and inhibition assays
-
Babon JJ, Murphy JM. In vitro JAK kinase activity and inhibition assays. Methods Mol Biol 2013; 967: 39-55.
-
(2013)
Methods Mol Biol
, vol.967
, pp. 39-55
-
-
Babon, J.J.1
Murphy, J.M.2
-
50
-
-
37349131080
-
Cytoplasmic p53 is not required for PUMA-induced apoptosis
-
Callus BA, Ekert PG, Heraud JE, Jabbour AM, Kotevski A, Vince JE et al. Cytoplasmic p53 is not required for PUMA-induced apoptosis. Cell Death Diff 2008; 15: 213-215.
-
(2008)
Cell Death Diff
, vol.15
, pp. 213-215
-
-
Callus, B.A.1
Ekert, P.G.2
Heraud, J.E.3
Jabbour, A.M.4
Kotevski, A.5
Vince, J.E.6
-
51
-
-
47549097367
-
TWEAK-FN14 signaling induces lysosomal degradation of a cIAP1-TRAF2 complex to sensitize tumor cells to TNFalpha
-
Vince JE, Chau D, Callus B, Wong WW, Hawkins CJ, Schneider P et al. TWEAK-FN14 signaling induces lysosomal degradation of a cIAP1-TRAF2 complex to sensitize tumor cells to TNFalpha. J Cell Biol 2008; 182: 171-184.
-
(2008)
J Cell Biol
, vol.182
, pp. 171-184
-
-
Vince, J.E.1
Chau, D.2
Callus, B.3
Wong, W.W.4
Hawkins, C.J.5
Schneider, P.6
-
52
-
-
0032480013
-
A new method for isolating tyrosine kinase substrates used to identify fish, an SH3 and PX domain-containing protein, and Src substrate
-
Lock P, Abram CL, Gibson T, Courtneidge SA. A new method for isolating tyrosine kinase substrates used to identify fish, an SH3 and PX domain-containing protein, and Src substrate. EMBO J 1998; 17: 4346-4357.
-
(1998)
EMBO J
, vol.17
, pp. 4346-4357
-
-
Lock, P.1
Abram, C.L.2
Gibson, T.3
Courtneidge, S.A.4
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