ATG16L1 and IL23R Variants and Genetic Susceptibility to Crohn's Disease: Mode of Inheritance Based on Meta-analysis of Genetic Association Studies

Inflammatory Bowel Diseases

Volumn 21, Issue 4, 2015, Pages 768-776

  Grigoras, Christos A ^a   Ziakas, Panayiotis D ^a   Jayamani, Elamparithi ^a   Mylonakis, Eleftherios ^a  

^a BROWN UNIVERSITY   (United States)

Author keywords

autophagy; Crohn's disease; interleukin; mode of inheritance

Indexed keywords

INTERLEUKIN 23 RECEPTOR; ATG16L1 PROTEIN, HUMAN; CARRIER PROTEIN; IL23R PROTEIN, HUMAN; INTERLEUKIN RECEPTOR;

ARTICLE; ASIAN; ATG16L1 GENE; CAUCASIAN; CROHN DISEASE; GENE; GENE FREQUENCY; GENETIC ASSOCIATION; GENETIC RISK; GENETIC SUSCEPTIBILITY; GENETIC VARIABILITY; GENOTYPE; HETEROZYGOSITY; HOMOZYGOTE; HUMAN; IL23R GENE; INHERITANCE; MEDLINE; POPULATION; PRIORITY JOURNAL; RISK ASSESSMENT; SINGLE NUCLEOTIDE POLYMORPHISM; SYSTEMATIC REVIEW; WEB OF SCIENCE; GENETIC PREDISPOSITION; GENETICS; META ANALYSIS; ODDS RATIO;

CARRIER PROTEINS; CROHN DISEASE; GENETIC ASSOCIATION STUDIES; GENETIC PREDISPOSITION TO DISEASE; GENETIC VARIATION; HUMANS; ODDS RATIO; RECEPTORS, INTERLEUKIN;

EID: 84925403090     PISSN: 10780998     EISSN: 15364844     Source Type: Journal    
DOI: 10.1097/MIB.0000000000000305     Document Type: Article

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