Genetic variants in IL-23R and ATG16L1 independently predispose to increased susceptibility to Crohn's disease in a Canadian population

Journal of Clinical Gastroenterology

Volumn 43, Issue 5, 2009, Pages 444-447

  Newman, William G ^a   Zhang, Qing ^b   Liu, Xiangdong ^c,d,e,f   Amos, Christopher I ^b   Siminovitch, Katherine A ^c,d,e,f  

^a UNIVERSITY OF MANCHESTER   (United Kingdom)

^b UNIVERSITY OF TEXAS MD ANDERSON CANCER CENTER   (United States)

^c UNIVERSITY OF TORONTO   (Canada)

^d MOUNT SINAI HOSPITAL   (Canada)

^e UNIVERSITY OF TORONTO   (Canada)

^f UNIVERSITY HEALTH NETWORK   (Canada)

Author keywords

Association; ATG16L1; Crohn's disease; IL 23 receptor; Inflammatory bowel disease; Single nucleotide polymorphisms

Indexed keywords

INTERLEUKIN 23 RECEPTOR;

ADULT; ALLELE; ARTICLE; ATG16L1 GENE; CANADA; CONTROLLED STUDY; CROHN DISEASE; ETHNICITY; FEMALE; GENE; GENETIC ASSOCIATION; GENETIC PREDISPOSITION; GENETIC RISK; GENETIC SUSCEPTIBILITY; GENETIC VARIABILITY; GENOTYPE; HAPLOTYPE; HOMOZYGOSITY; HUMAN; MAJOR CLINICAL STUDY; MALE; PATHOGENESIS; PHENOTYPE; PRIORITY JOURNAL; RISK ASSESSMENT; SINGLE NUCLEOTIDE POLYMORPHISM; ULCERATIVE COLITIS;

CANADA; CARRIER PROTEINS; CASE-CONTROL STUDIES; COLITIS, ULCERATIVE; CROHN DISEASE; EUROPEAN CONTINENTAL ANCESTRY GROUP; GENE FREQUENCY; GENETIC PREDISPOSITION TO DISEASE; HUMANS; JEWS; LOGISTIC MODELS; ODDS RATIO; POLYMORPHISM, SINGLE NUCLEOTIDE; RECEPTORS, INTERLEUKIN; RISK ASSESSMENT; RISK FACTORS;

EID: 67651095721     PISSN: 01920790     EISSN: None     Source Type: Journal    
DOI: 10.1097/MCG.0b013e318168bdf0     Document Type: Article

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