Bcl-2 family proteins in breast development and cancer: Could Mcl-1 targeting overcome therapeutic resistance?

Oncotarget

Volumn 6, Issue 6, 2015, Pages 3519-3530

  Williams, Michelle M ^a   Cook, Rebecca S ^a,b  

^a VANDERBILT UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b VANDERBILT INGRAM CANCER CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DOXORUBICIN; GEMCITABINE; NAVITOCLAX; PACLITAXEL; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2; PROTEIN MCL 1; SORAFENIB; TAMOXIFEN; TAXANE DERIVATIVE; TRASTUZUMAB;

APOPTOSIS; ARTICLE; BREAST CANCER; BREAST CARCINOGENESIS; CANCER GROWTH; CANCER RESISTANCE; CELL KILLING; CELL PROLIFERATION; CELL SURVIVAL; DOWN REGULATION; DRUG EFFICACY; DRUG RESPONSE; DRUG SAFETY; DRUG TOLERABILITY; HER2 GENE; HUMAN; NONHUMAN; ONCOGENE; PROTEIN EXPRESSION; PROTEIN FAMILY; PROTEIN FUNCTION; PROTEIN LOCALIZATION; PROTEIN PROTEIN INTERACTION; PROTEIN TARGETING; UPREGULATION; ANIMAL; BREAST; BREAST TUMOR; CYTOLOGY; FEMALE; GENETICS; GROWTH, DEVELOPMENT AND AGING; METABOLISM; PATHOLOGY; PHYSIOLOGY; UDDER;

ANIMALS; APOPTOSIS; BREAST; BREAST NEOPLASMS; FEMALE; HUMANS; MAMMARY GLANDS, ANIMAL; MYELOID CELL LEUKEMIA SEQUENCE 1 PROTEIN; PROTO-ONCOGENE PROTEINS C-BCL-2;

EID: 84924229796     PISSN: None     EISSN: 19492553     Source Type: Journal    
DOI: 10.18632/oncotarget.2792     Document Type: Article

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