Pathophysiological functions of the CagA oncoprotein during infection by Helicobacter pylori

Microbes and Infection

Volumn 13, Issue 10, 2011, Pages 799-807

  Murata Kamiya, Naoko ^a  

^a UNIVERSITY OF TOKYO   (Japan)

Author keywords

CagA; Gastric carcinoma; Helicobacter pylori

Indexed keywords

ACTIVATION INDUCED CYTIDINE DEAMINASE; BETA CATENIN; C TERMINAL SRC KINASE; CAGA PROTEIN; PARTITIONING DEFECTIVE 1; PHOSPHOTRANSFERASE; PROTEIN TYROSINE PHOSPHATASE SHP 2; UNCLASSIFIED DRUG;

BACTERIAL GENE; BACTERIAL STRAIN; BACTERIAL TRANSLOCATION; CARBOXY TERMINAL SEQUENCE; CELL DISRUPTION; CELL GROWTH; CELL MOTILITY; CELL POLARITY; DEREGULATION; ENZYME INDUCTION; EPITHELIUM CELL; EXPERIMENTAL MODEL; HELICOBACTER INFECTION; HELICOBACTER PYLORI; HOST CELL; HUMAN; MITOSIS; NONHUMAN; ONCOGENE; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN INTERACTION; PROTEIN MULTIMERIZATION; PROTEIN PHOSPHORYLATION; SHORT SURVEY; SRC HOMOLOGY DOMAIN; STOMACH CARCINOMA; STOMACH EPITHELIUM; TUMOR SUPPRESSOR GENE;

ANTIGENS, BACTERIAL; BACTERIAL PROTEINS; CARCINOMA; HELICOBACTER INFECTIONS; HELICOBACTER PYLORI; HUMANS; MODELS, BIOLOGICAL; ONCOGENE PROTEINS; STOMACH NEOPLASMS; VIRULENCE FACTORS;

HELICOBACTER PYLORI;

EID: 79960840217     PISSN: 12864579     EISSN: 1769714X     Source Type: Journal    
DOI: 10.1016/j.micinf.2011.03.011     Document Type: Short Survey

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