Constitutive expression of AID leads to tumorigenesis

Journal of Experimental Medicine

Volumn 197, Issue 9, 2003, Pages 1173-1181

  Okazaki, Il Mi ^a   Hiai, Hiroshi ^a   Kakazu, Naoki ^b   Yamada, Shuichi ^a   Muramatsu, Masamichi ^a   Kinoshita, Kazuo ^a   Honjo, Tasuku ^a  

^a KYOTO UNIVERSITY   (Japan)

^b KYOTO PREFECTURAL UNIVERSITY OF MEDICINE   (Japan)

Author keywords

Class switch recombination; Genomic instability; Lymphoma; Somatic hypermutation; T cell receptor gene

Indexed keywords

CYTIDINE DEAMINASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; B LYMPHOCYTE; BRONCHIOLE; CARCINOGENESIS; CHROMOSOME MUTATION; CHROMOSOME TRANSLOCATION; CONTROLLED STUDY; DNA RECOMBINATION; DNA REPAIR; ENZYME ACTIVATION; FEMALE; FIBROBLAST; GENE MUTATION; GENE REARRANGEMENT; GENE TRANSLOCATION; GENOME; IMMUNOGLOBULIN GENE; LYMPHOCYTE; LYMPHOMA CELL; MALE; MOUSE; NONHUMAN; ONCOGENE C MYC; POINT MUTATION; PRIORITY JOURNAL; PROTEIN EXPRESSION; REGULATORY MECHANISM; RESPIRATORY EPITHELIUM; T CELL LYMPHOMA; T LYMPHOCYTE RECEPTOR GENE; TRANSGENIC MOUSE;

ANIMALS; BASE SEQUENCE; BLOTTING, SOUTHERN; CELL TRANSFORMATION, NEOPLASTIC; CYTIDINE DEAMINASE; DNA PRIMERS; FLOW CYTOMETRY; LYMPHOID TISSUE; MICE; MICE, INBRED C57BL; MICE, SCID; MICE, TRANSGENIC; POINT MUTATION; RECEPTORS, ANTIGEN, T-CELL; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;

EID: 0037881911     PISSN: 00221007     EISSN: None     Source Type: Journal    
DOI: 10.1084/jem.20030275     Document Type: Article

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