Increased expression of bim contributes to the potentiation of serum deprivation-induced apoptotic cell death in huntington's disease knock-in striatal cell line

Neurological Research

Volumn 31, Issue 1, 2009, Pages 77-83

  Kong, Pil Jae ^a   Kil, Myung Og ^a   Lee, Heejae ^a   Kim, Sung Soo ^a   Johnson, Gail V W ^b   Chun, Wanjoo ^a  

^a Kangwon National University   (South Korea)

^b UNIVERSITY OF ROCHESTER MEDICAL CENTER   (United States)

Author keywords

Akt; Apoptosis; Bim; Huntington's disease; Serum deprivation; Striatal neuron

Indexed keywords

BIM PROTEIN; HUNTINGTIN; PROTEIN BCL 2; PROTEIN KINASE B; SOMATOMEDIN C; TRANSCRIPTION FACTOR FKHRL1;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; ATTENUATION; BRAIN NERVE CELL; CELL DEATH; CELL IMMORTALIZATION; CELL LINE; CELL PROTECTION; COMPARATIVE STUDY; CONTROLLED STUDY; CORPUS STRIATUM; EMBRYO; HUNTINGTON CHOREA; MOUSE; MUTANT; NONHUMAN; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; SERUM; WILD TYPE;

ANIMALS; APOPTOSIS; APOPTOSIS REGULATORY PROTEINS; BLOTTING, WESTERN; CELL LINE; CORPUS STRIATUM; FORKHEAD TRANSCRIPTION FACTORS; GENE EXPRESSION; GENE KNOCK-IN TECHNIQUES; HUNTINGTON DISEASE; MEMBRANE PROTEINS; MICE; NERVE TISSUE PROTEINS; NEURONS; NUCLEAR PROTEINS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-AKT;

EID: 60849125108     PISSN: 01616412     EISSN: None     Source Type: Journal    
DOI: 10.1179/174313208X331572     Document Type: Article

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