Sustained oxidative stress inhibits NF-κB activation partially via inactivating the proteasome

Free Radical Biology and Medicine

Volumn 46, Issue 1, 2009, Pages 62-69

  Wu, Mingxing ^a,b   Bian, Qingning ^a,b   Liu, Yizhi ^a   Fernandes, Alexandre F ^b,c   Taylor, Allen ^b   Pereira, Paulo ^c   Shang, Fu ^b  

^a SUN YAT SEN UNIVERSITY   (China)

^b TUFTS UNIVERSITY   (United States)

^c UNIVERSITY OF COIMBRA   (Portugal)

Author keywords

Free radicals; Lens; NF B; Oxidative stress; Proteasome; Signal transduction

Indexed keywords

HYDROGEN PEROXIDE; I KAPPA B; I KAPPA B ALPHA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; PROTEASOME; TUMOR NECROSIS FACTOR ALPHA;

ARTICLE; CELL DEATH; CELL VIABILITY; CONTROLLED STUDY; ENZYME ACTIVITY; EPITHELIUM CELL; HUMAN; HUMAN CELL; LENS EPITHELIUM; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN PHOSPHORYLATION;

CATARACT; CELL SURVIVAL; EPITHELIAL CELLS; HUMANS; HYDROGEN PEROXIDE; I-KAPPA B PROTEINS; LENS, CRYSTALLINE; NF-KAPPA B; OXIDATIVE STRESS; PHOSPHORYLATION; PROTEASOME ENDOPEPTIDASE COMPLEX; SIGNAL TRANSDUCTION; TIME FACTORS; TRANSCRIPTIONAL ACTIVATION; TUMOR NECROSIS FACTOR-ALPHA;

EID: 57449104348     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.freeradbiomed.2008.09.021     Document Type: Article

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