Vitamin A depletion causes oxidative stress, mitochondrial dysfunction, and PARP-1-dependent energy deprivation

FASEB Journal

Volumn 22, Issue 11, 2008, Pages 3878-3887

  Chiu, Haw Jyh ^a   Fischman, Donald A ^b   Hammerling, Ulrich ^a  

^a MEMORIAL SLOAN KETTERING CANCER CENTER   (United States)

^b WEILL CORNELL MEDICAL COLLEGE   (United States)

Author keywords

Bioenergetics; Lymphocytes; Mitochondrion; Reactive oxygen species

Indexed keywords

ADENOSINE TRIPHOSPHATE; CASPASE 3; ENDONUCLEASE; NICOTINAMIDE ADENINE DINUCLEOTIDE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE 1; REACTIVE OXYGEN METABOLITE; RETINOL;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; C57BL 6 MOUSE; CELL DEATH; CHROMATIN CONDENSATION; DEPLETION; DISORDERS OF MITOCHONDRIAL FUNCTIONS; DNA DEGRADATION; ENERGY CONSUMPTION; ENZYME ACTIVATION; HOMEOSTASIS; HYPOXIA; LEUKEMIA CELL LINE; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; T LYMPHOCYTE; VITAMIN A DEPLETION;

ADENOSINE TRIPHOSPHATE; ANIMALS; APOPTOSIS; CELL HYPOXIA; DNA FRAGMENTATION; ENERGY METABOLISM; ENZYME ACTIVATION; HOMEOSTASIS; HUMANS; JURKAT CELLS; MICE; MICE, KNOCKOUT; MITOCHONDRIA; NAD; OXIDATIVE STRESS; POLY(ADP-RIBOSE) POLYMERASES; REACTIVE OXYGEN SPECIES; VITAMIN A; VITAMIN A DEFICIENCY;

EID: 55549139488     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.08-112375     Document Type: Article

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