Defective responses to DNA single- and double-strand breaks in spinocerebellar ataxia

DNA Repair

Volumn 7, Issue 7, 2008, Pages 1061-1076

  Lavin, Martin F ^a,b   Gueven, Nuri ^a   Grattan Smith, Padraic ^c  

^a QIMR BERGHOFER MEDICAL RESEARCH INSTITUTE   (Australia)

^b UNIVERSITY OF QUEENSLAND   (Australia)

^c SYDNEY CHILDREN'S HOSPITAL   (Australia)

Author keywords

DNA damage response; Genome instability; Neurodegeneration; Spinocerebellar ataxia

Indexed keywords

ARTICLE; ATAXIA TELANGIECTASIA; CANCER RISK; CELL CYCLE S PHASE; CHROMATIN; DEGENERATIVE DISEASE; DNA DAMAGE; DNA REPAIR; DNA STRAND BREAKAGE; GENOMIC INSTABILITY; MITOSIS; NERVE DEGENERATION; OXIDATIVE STRESS; PHENOTYPE; PRIORITY JOURNAL; SPINOCEREBELLAR DEGENERATION;

APOPTOSIS; ATAXIA TELANGIECTASIA; DNA; DNA BREAKS, DOUBLE-STRANDED; DNA BREAKS, SINGLE-STRANDED; DNA DAMAGE; DNA, SINGLE-STRANDED; HUMANS; PHENOTYPE; S PHASE; SPINOCEREBELLAR DEGENERATIONS;

EID: 44949234489     PISSN: 15687864     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.dnarep.2008.03.008     Document Type: Article

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