Apoptosis induced by replication inhibitors in Chk1-depleted cells is dependent upon the helicase cofactor Cdc45

Cell Death and Differentiation

Volumn 15, Issue 5, 2008, Pages 889-898

  Rodriguez, R ^a   Gagou, M E ^a   Meuth, M ^a  

^a UNIVERSITY OF SHEFFIELD   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

CELL CYCLE PROTEIN; CHECKPOINT KINASE 1; HELICASE; HELICASE COFACTOR; PROTEIN CDC45; PROTEIN P21; REPLICATION FACTOR A; SINGLE STRANDED DNA; SMALL INTERFERING RNA; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; CANCER CELL CULTURE; CELL CYCLE S PHASE; CELL FREE SYSTEM; CELL PROTECTION; CELL VIABILITY; COLON CANCER; COMPLEX FORMATION; CONTROLLED STUDY; DNA REPLICATION; DNA REPLICATION ORIGIN; DNA REPLICATION TIMING; DNA SYNTHESIS INHIBITION; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROTEIN ASSEMBLY; PROTEIN DEPLETION; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION; TRANSLATION REGULATION;

ANIMALS; APOPTOSIS; CELL CYCLE; CELL CYCLE PROTEINS; CELL LINE; CYCLIN-DEPENDENT KINASE INHIBITOR P21; DNA REPLICATION; DNA, SINGLE-STRANDED; HUMANS; PROTEIN KINASES; REPLICATION PROTEIN A; RNA, SMALL INTERFERING;

EID: 42149152648     PISSN: 13509047     EISSN: 14765403     Source Type: Journal    
DOI: 10.1038/cdd.2008.4     Document Type: Article

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