Deficiencies of double-strand break repair factors and effects on mutagenesis in directly γ-irradiated and medium-mediated bystander human lymphoblastoid cells

Radiation Research

Volumn 169, Issue 2, 2008, Pages 197-206

  Zhang, Ying ^a   Zhou, Junqing ^a   Held, Kathryn D ^b   Redmond, Robert W ^b   Prise, Kevin M ^c   Liber, Howard L ^a,d  

^a Department of Environmental and Radiological Health Sciences

^b MASSACHUSETTS GENERAL HOSPITAL   (United States)

^c QUEEN'S UNIVERSITY BELFAST   (United Kingdom)

^d UNIVERSITY OF COLORADO CANCER CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CATALASE; HYDROGEN PEROXIDE; PROTEIN P53; RAD54 PROTEIN; THYMIDINE KINASE;

ARTICLE; BYSTANDER EFFECT; CELL LINE; CELL MUTANT; DNA REPAIR; DOUBLE STRANDED DNA BREAK; GAMMA IRRADIATION; HOMOLOGOUS RECOMBINATION; HUMAN; HUMAN CELL; LYMPHOBLASTOID CELL; MUTAGENESIS; PRIORITY JOURNAL; RADIATION; RNA INTERFERENCE; WILD TYPE;

BYSTANDER EFFECT; CELL LINE; CULTURE MEDIA, CONDITIONED; DNA BREAKS, DOUBLE-STRANDED; DNA REPAIR; DOSE-RESPONSE RELATIONSHIP, RADIATION; GAMMA RAYS; HUMANS; LYMPHOCYTES; MUTAGENESIS; RADIATION DOSAGE; TUMOR SUPPRESSOR PROTEIN P53;

EID: 39749107193     PISSN: 00337587     EISSN: None     Source Type: Journal    
DOI: 10.1667/RR1189.1     Document Type: Article

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