Physiologically regulated transgenic ABCA1 does not reduce amyloid burden or amyloid-β peptide levels in vivo

Journal of Lipid Research

Volumn 48, Issue 4, 2007, Pages 914-923

  Hirsch Reinshagen, Veronica ^a   Chan, Jennifer Y ^a   Wilkinson, Anna ^a   Tanaka, Tracie ^a   Fan, Jianjia ^a   Ou, George ^a   Maia, Luis F ^b   Singaraja, Roshni R ^a   Hayden, Michael R ^a   Wellington, Cheryl L ^a  

^a UNIVERSITY OF BRITISH COLUMBIA   (Canada)

^b HOSPITAL DE SANTO ANTÓNIO   (Portugal)

Author keywords

Alzheimer's disease; Animal model; Apolipoprotein E; ATP binding cassette transporter A1

Indexed keywords

ABC TRANSPORTER A1; AMYLOID; AMYLOID BETA PROTEIN; APOLIPOPROTEIN E; BRAIN PROTEIN; MESSENGER RNA;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BACTERIAL ARTIFICIAL CHROMOSOME; BRAIN CORTEX; CEREBROSPINAL FLUID; GENE OVEREXPRESSION; GENETIC ANALYSIS; LIPID ANALYSIS; LIPID STORAGE; MOUSE; NEUROPATHOLOGY; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN EXPRESSION; PROTEIN FUNCTION; TRANSCRIPTION REGULATION;

AGE FACTORS; AMYLOID; AMYLOID BETA-PROTEIN; ANIMALS; APOLIPOPROTEINS E; ATP-BINDING CASSETTE TRANSPORTERS; BRAIN CHEMISTRY; CEREBELLUM; CEREBRAL CORTEX; CHROMOSOMES, ARTIFICIAL, BACTERIAL; MICE; MICE, TRANSGENIC; RNA, MESSENGER;

EID: 34147169863     PISSN: 00222275     EISSN: 15397262     Source Type: Journal    
DOI: 10.1194/jlr.M600543-JLR200     Document Type: Article

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