Axonal protection achieved in a model of multiple sclerosis using lamotrigine

Journal of Neurology

Volumn 253, Issue 12, 2006, Pages 1542-1551

  Bechtold, David A ^a   Miller, Sandra J ^a   Dawson, Angela C ^a   Sun, Yue ^a   Kapoor, Raju ^a,b   Berry, David ^c   Smith, Kenneth J ^a  

^a KING'S COLLEGE LONDON   (United Kingdom)

^b NATIONAL HOSPITAL FOR NEUROLOGY AND NEUROSURGERY   (United Kingdom)

^c GUY'S AND ST THOMAS' NHS FOUNDATION TRUST   (United Kingdom)

Author keywords

Axonal loss; Degeneration; EAE; Sodium channel

Indexed keywords

LAMOTRIGINE; SODIUM CHANNEL BLOCKING AGENT;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DISEASE MODEL; ENCEPHALOMYELITIS; HISTOLOGY; INOCULATION; MALE; MULTIPLE SCLEROSIS; NERVE FIBER; NERVE FIBER DEGENERATION; NERVE FUNCTION; NERVE POTENTIAL; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; RAT; SPINAL CORD; THORACIC SPINE;

ACTION POTENTIALS; ANIMALS; DISEASE MODELS, ANIMAL; DOSE-RESPONSE RELATIONSHIP, DRUG; MALE; MULTIPLE SCLEROSIS; NERVE DEGENERATION; NEUROPROTECTIVE AGENTS; RATS; SPINAL CORD; STATISTICS, NONPARAMETRIC; TIME FACTORS; TRIAZINES;

EID: 33744489487     PISSN: 03405354     EISSN: 14321459     Source Type: Journal    
DOI: 10.1007/s00415-006-0204-1     Document Type: Article

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