Axonal protection in experimental autoimmune neuritis by the sodium channel blocking agent flecainide

Brain

Volumn 128, Issue 1, 2005, Pages 18-28

  Bechtold, David A ^a   Yue, Xu ^a   Evans, Richard M ^a   Davies, Meirion ^a   Gregson, Norman A ^a   Smith, Kenneth J ^a  

^a KING'S COLLEGE LONDON   (United Kingdom)

Author keywords

Axonal degeneration; EAN; GBS; Guillain Barr syndrome; Neuroinflammation

Indexed keywords

FLECAINIDE ACETATE; NITRIC OXIDE; SODIUM CHANNEL BLOCKING AGENT;

ACUTE DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOIMMUNE DISEASE; CELL SURVIVAL; CONTROLLED STUDY; DEMYELINATING NEUROPATHY; DISABILITY; DRUG EFFECT; EXPERIMENTAL AUTOIMMUNE NEURITIS; FEMALE; GUILLAIN BARRE SYNDROME; HISTOLOGY; NERVE FIBER; NERVE FIBER DEGENERATION; NEURITIS; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; RAT; TIBIAL NERVE;

ANIMALS; ANTIBODIES; AXONS; CELL COUNT; ELECTROPHYSIOLOGY; FEMALE; FLECAINIDE; INJECTIONS, SUBCUTANEOUS; MACROPHAGES; MYELIN SHEATH; NERVE DEGENERATION; NEURITIS, AUTOIMMUNE, EXPERIMENTAL; RATS; RATS, INBRED LEW; SODIUM CHANNEL BLOCKERS; TIBIAL NERVE;

EID: 11244260444     PISSN: 00068950     EISSN: None     Source Type: Journal    
DOI: 10.1093/brain/awh328     Document Type: Article

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