Mitochondrial dysfunction in T cells of patients with systemic lupus erythematosus

International Reviews of Immunology

Volumn 23, Issue 3-4, 2004, Pages 293-313

  Perl, Andras ^a   Gergely Jr , Peter ^b   Banki, Katalin ^b  

^a SUNY UPSTATE MEDICAL UNIVERSITY   (United States)

^b State University of New York Upstate Medical University: College of Medicine   (United States)

Author keywords

Human; Lupus; Signal transduction

Indexed keywords

ADENOSINE TRIPHOSPHATE; CASPASE; GLUTATHIONE; REACTIVE OXYGEN METABOLITE;

ALKALINIZATION; APOPTOSIS; CELL DEATH; CELL MEMBRANE POTENTIAL; CELL SURVIVAL; CYTOPLASM; DISORDERS OF MITOCHONDRIAL FUNCTIONS; DRUG TARGETING; ENZYME ACTIVITY; HUMAN; HYPERPOLARIZATION; LYMPHOCYTE PROLIFERATION; MITOCHONDRIAL MEMBRANE; OXIDATION REDUCTION REACTION; PRIORITY JOURNAL; REVIEW; SYSTEMIC LUPUS ERYTHEMATOSUS; T LYMPHOCYTE; T LYMPHOCYTE ACTIVATION;

ADENOSINE TRIPHOSPHATE; APOPTOSIS; HUMANS; INTERLEUKIN-10; INTERLEUKIN-12; LUPUS ERYTHEMATOSUS, SYSTEMIC; LYMPHOCYTE ACTIVATION; MEMBRANE POTENTIALS; MITOCHONDRIA; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION; T-LYMPHOCYTES;

EID: 3042617625     PISSN: 08830185     EISSN: None     Source Type: Journal    
DOI: 10.1080/08830180490452576     Document Type: Review

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