Regulation of endoplasmic reticulum Ca2+ dynamics by proapoptotic BCL-2 family members

Biochemical Pharmacology

Volumn 66, Issue 8, 2003, Pages 1335-1340

  Oakes, Scott A ^a,b   Opferman, Joseph T ^a   Pozzan, Tullio ^c   Korsmeyer, Stanley J ^a   Scorrano, Luca ^d  

^a HOWARD HUGHES MEDICAL INSTITUTE   (United States)

^b BRIGHAM AND WOMEN S HOSPITAL   (United States)

^c UNIVERSITY OF PADOVA   (Italy)

^d VENETIAN INSTITUTE OF MOLECULAR MEDICINE   (Italy)

Author keywords

Bax, Bak double knockouts; DKO; Endoplasmic reticulum; ER; Inositol 1,4,5 trisphosphate; IP3; Mitochondrial permeability transition; Mitochondrially targeted BAX; mtBAX; Permeability transition pore; PT; PTP; SERCA

Indexed keywords

CALCIUM; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2; STAUROSPORINE; BAK1 PROTEIN, HUMAN; BAX PROTEIN, HUMAN; MEMBRANE PROTEIN; ONCOPROTEIN;

APOPTOSIS; CALCIUM CELL LEVEL; CELL DEATH; CELL STIMULATION; CONFERENCE PAPER; ENDOPLASMIC RETICULUM; GENE OVEREXPRESSION; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; STEADY STATE; ANIMAL; CALCIUM SIGNALING; HUMAN; METABOLISM; MITOCHONDRION; REVIEW;

ANIMALS; APOPTOSIS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; CALCIUM; CALCIUM SIGNALING; ENDOPLASMIC RETICULUM; HUMANS; MEMBRANE PROTEINS; MITOCHONDRIA; OXIDATIVE STRESS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2;

EID: 0642345210     PISSN: 00062952     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0006-2952(03)00482-9     Document Type: Conference Paper

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