Bloom syndrome cells undergo p53-dependent apoptosis and delayed assembly of BRCA1 and NBS1 repair complexes at stalled replication forks

Journal of Cell Biology

Volumn 162, Issue 7, 2003, Pages 1197-1209

  Davalos, Albert R ^a   Campisi, Judith ^a,b  

^a LAWRENCE BERKELEY NATIONAL LABORATORY   (United States)

^b BUCK INSTITUTE FOR RESEARCH ON AGING   (United States)

Author keywords

H2AX; DNA damage; DNA repair; DNA replication; S phase checkpoint

Indexed keywords

BRCA1 PROTEIN; HELICASE; NIBRIN; PROTEIN; PROTEIN P53; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; BLM GENE; BLOOM SYNDROME; CATALYSIS; CELL KINETICS; CONTROLLED STUDY; DNA DAMAGE; DNA REPLICATION; FIBROBLAST; GENE; GENETIC CODE; GENETIC STABILITY; GROWTH RETARDATION; HEREDITY; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROTEIN ASSEMBLY; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION;

ADENOSINE TRIPHOSPHATASES; APOPTOSIS; BLOOM SYNDROME; BRCA1 PROTEIN; CELL CYCLE PROTEINS; CELL DIVISION; CELL LINE, TRANSFORMED; CELLS, CULTURED; DNA HELICASES; DNA REPAIR; DNA REPLICATION; DNA-BINDING PROTEINS; ETOPOSIDE; FIBROBLASTS; G1 PHASE; HUMANS; NUCLEAR PROTEINS; NUCLEIC ACID SYNTHESIS INHIBITORS; PHENOTYPE; S PHASE; TELOMERASE; TUMOR SUPPRESSOR PROTEIN P53;

EID: 0141864666     PISSN: 00219525     EISSN: None     Source Type: Journal    
DOI: 10.1083/jcb.200304016     Document Type: Article

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