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Volumn 21, Issue 14, 2002, Pages 2141-2153
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Ganciclovir-induced apoptosis in HSV-1 thymidine kinase expressing cells: Critical role of DNA breaks, Bcl-2 decline and caspase-9 activation
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Author keywords
Apoptosis; Bcl 2 cleavage; Caspase 9; DNA breakage; Ganciclovir; Suicide gene therapy
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Indexed keywords
CASPASE 3;
CASPASE 8;
CASPASE 9;
CASPASE INHIBITOR;
CYTOCHROME C;
DOUBLE STRANDED DNA;
FAS ANTIGEN;
GANCICLOVIR;
PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR;
PROTEIN BAD;
PROTEIN BCL 2;
PROTEIN P53;
THYMIDINE KINASE;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
CELL CYCLE G2 PHASE;
CELL CYCLE S PHASE;
CHO CELL;
CONTROLLED STUDY;
DEPHOSPHORYLATION;
DNA DEGRADATION;
DNA STRAND BREAKAGE;
ENZYME ACTIVATION;
EXPRESSION VECTOR;
HERPES SIMPLEX VIRUS 1;
MEMBRANE DAMAGE;
MITOCHONDRIAL MEMBRANE;
MITOSIS INHIBITION;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN DEGRADATION;
SUICIDE GENE THERAPY;
ANIMALS;
ANTIGENS, CD95;
APOPTOSIS;
CASPASE 9;
CASPASES;
CELL CYCLE;
CHO CELLS;
CRICETINAE;
CYTOCHROME C GROUP;
DNA DAMAGE;
ENZYME ACTIVATION;
GANCICLOVIR;
GENE EXPRESSION REGULATION, NEOPLASTIC;
HERPESVIRUS 1, HUMAN;
POLY(ADP-RIBOSE) POLYMERASES;
PROTO-ONCOGENE PROTEINS C-BCL-2;
REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;
THYMIDINE KINASE;
TIME FACTORS;
TRANSFECTION;
ANIMALIA;
HUMAN HERPESVIRUS 1;
SIMPLEXVIRUS;
VECTORS;
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EID: 0037192631
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1205280 Document Type: Article |
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Times cited : (104)
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References (53)
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