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Volumn 17, Issue 9, 1997, Pages 3064-3073

Mechanisms of cell death induced by the mitochondrial toxin 3- nitropropionic acid: Acute excitotoxic necrosis and delayed apoptosis

Author keywords

3 nitropropionic acid; apoptosis; ATP; energy metabolism; excitotoxicity; necrosis; nuclear fragmentation; succinate dehydrogenase; TUNEL

Indexed keywords

3 NITROPROPIONIC ACID; ADENOSINE TRIPHOSPHATE; CYCLOHEXIMIDE; DIZOCILPINE; EXCITOTOXIN; GLUTAMIC ACID; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; N METHYL DEXTRO ASPARTIC ACID RECEPTOR BLOCKING AGENT; SUCCINATE DEHYDROGENASE;

EID: 0030946503     PISSN: 02706474     EISSN: None     Source Type: Journal    
DOI: 10.1523/jneurosci.17-09-03064.1997     Document Type: Article
Times cited : (218)

References (10)
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  • 9
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    • Apoptosis and necrosis: Two distinct events induced, respectively, by mild and intense insults with N-methyl-D-aspartate or nitric oxide/ superoxide in cortical cell cultures
    • Bonfoco E, Krainc D, Ankarcrona M, Nicotera P, Lipton SA (1995) Apoptosis and necrosis: two distinct events induced, respectively, by mild and intense insults with N-methyl-D-aspartate or nitric oxide/ superoxide in cortical cell cultures. Proc Natl Acad Sci USA 92:7162-7166.
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    • Bonfoco, E.1    Krainc, D.2    Ankarcrona, M.3    Nicotera, P.4    Lipton, S.A.5


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.